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the spread of damage to the neighborhood.
Our study has shown that the heavy metal ions by mimicking calcium not only evade the boundary frisking but also bind to the calcium-binding region of the scramblase protein, resulting in the activation of the membrane proteins. The heavy metal pollutants activate the scramblases similar to calcium ions but at very low concentrations. Once activated, the scramblases initiate the flipping of charged lipids, thereby raising the alarms for the removal of damaged cells. Removal of infected or infested
cells prevents the spread of further damage, saving the entire neighborhood from a broad infestation.
This small membrane protein is one of our lifesavers and the peace-keeping forces of our defense system. As the saying goes, the size of the mustard does not determine its spiciness. These relatively small membrane proteins are a major player in our elaborate defense system.
Nature has its unique way of flushing toxins and their infestation from our clean system, which ranges from our small cell to a big ocean to our broad universe.
Scramblases Calcium-dependent Pushes the charged lipids out Rings the death bells oft.
Mr. Santoshkumar Palanirajan || 509
My work focuses on what really happens inside the cells after the attack by heavy metals such as lead and mercury. We are working on a small protein molecule called “scramblase,” anchored to this fluidic membrane. Scramblase, a membrane protein, plays the lead role in our story. Scramblases help in identifying and clearing the infected cells by initiating the response to several incoming attacks.