Page 125 - Consciousness in the Cell

HARUN YAHYA

THE DETAILS OF EVEN THE TINIEST MOLECULE

ARE ENOUGH TO DESTROY THE

THEORY OF EVOLUTION

The protein thrombin allows the blood to clot by converting fib-
rinogen to fibrin. Even though this protein is always circulating
through the blood, however, it doesn't always lead to the clotting
of blood, thereby preventing blood flow. If there is bleeding in one
blood vessel only, it "understands" that clotting is needed there
and sets about clotting the blood. If thrombin always led to clot-
ting, all of the blood in the vessels would clot as a result of the
thrombin present, and the body could not survive with its circula-
tion totally blocked. So how does thrombin attain the ability to
cause clotting only in the place where it's needed?
Thrombin is usually present in blood plasma in an inactive
form called prothrombin. Being inactive, it cannot perform the
duty of clotting and in this way we are protected from the fatal
consequences of uncontrolled clotting.
But what activates prothrombin and converts it to the clotting
factor thrombin when bleeding occurs?
A protein in the blood called Stuart factor has the effect of con-
verting prothrombin to thrombin. However Stuart factor is itself
also present in an inactive form, and needs to be activated.
At this point we are faced with a chicken-and-egg scenario. Still
another protein called accelerin is needed before Stuart factor can
become active and turn prothrombin into thrombin—thereby stop-
ping any bleeding in the organism. But what's truly remarkable is
that accelerin, too, is present in an inactive form of its own, called
proaccelerin. And what activates proaccelerin?
Thrombin!

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