Page 119 - Problem-Based Feline Medicine
P. 119

8 – THE CYANOTIC CAT  111



            TRAUMA
                       ● Diaphragmatic hernia* (p 115)
                       Dyspnea and tachypnea may occur acutely or months or years following blunt trauma. Vomiting,
                       regurgitation, inappetence and weight loss may also occur.

                       ● Central nervous system damage to the respiratory center (p 118)
                       Persistent or episodic weakness, Cheyne–Stokes respiration and cyanosis.
            TOXIC

                       ● Drug overdosage (p 117)
                       Excessive administration of barbiturates, narcotics, opiates, tranquilizers or anesthetic agents may
                       lead to decreased respiratory effort, loss of consciousness and cyanosis. During anesthesia,
                       reduced bleeding at surgical sites and indicators of a deep plane of anesthesia are present.
            PERIPHERAL HYPOXIA
            WHERE? – GENERALIZED
            MECHANICAL

                       ● Vasoconstriction* (p 118)
                       Peripheral hypoxia occurs due to vasoconstriction as a consequence of hypovolemic, cardiogenic
                       or septic shock or due to hypothermia. Clinical signs, physical findings and history point to one of
                       these causes. There may be evidence of shock, trauma, dehydration, pyrexia, blood loss or reduced
                       urinary output. Signs of cardiac failure include dyspnea, cardiac murmur, gallop rhythm, slow cap-
                       illary refill time, pale/cyanotic mucous membranes and pulse deficits. Frequent or severe vomiting,
                       diarrhea or diuresis may indicate fluid loss.

            LOCALIZED
            MECHANICAL
                       ● Arterial thromboembolism** (p 120)
                       Ninety percent lodge at the distal aortic bifurcation causing bilateral hindlimb pain and paresis, weak
                       or absent femoral pulses, cold, cyanotic footpads and lack of spontaneous movement of the muscles of
                       the hindlimbs. Other areas embolized less commonly are the thoracic aorta, renal, mesenteric, coro-
                       nary, cerebral and branchial arteries which cause acute, severe and variable clinical signs.

                       ● Venous obstruction (p 121)
                       Clinical signs relate to the area drained by the obstructed vein, mainly edema and vasodilatation
                       distal to the site of obstruction. Cranial vena caval obstruction results in edema of the neck, ventral
                       head, cranial thorax and forelimbs.
            ABNORMAL HEMOGLOBIN WITH DECREASED OXYGEN-CARRYING CAPACITY
            WHERE? – HEMOGLOBIN IN RED BLOOD CELLS
            WHAT?

            TOXICITY
                       ● Paracetamol/acetaminophen toxicity (p 121)
                       Acute onset and rapid progression of salivation, vomiting and depression. Cyanosis develops
                       within 4–12 hours of ingestion. Subcutaneous edema of the face may be evident, which may be
                       accompanied by lacrimation and puritis. Hematuria, hemolysis and Heinz bodies occur associated
                       with methemoglobin formation.
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