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The Ādam Paradox Hypothesis 161
BNC2 — Sensorimotor Integration (~200 ka)
A highly selected human gene linked to skin and neural integration (Sulem et al.,
2008).
Evolutionary gap: heightened integration, no symbolic imprint.
APH view: pre-installed to coordinate body-sense with symbolic gesture.
ANKRD11 — Cognition Tuning (~200 ka)
A neurodevelopmental regulator; mutations linked to cognitive syndromes (Lo-
Castro et al., 2013).
Evolutionary gap: regulatory tuning present, symbolic cognition absent.
APH view: resilience scaffold, activated fully at threshold.
Y-Chromosomal Bottleneck (~70 ka)
Y-CT lineage emerges post-bottleneck (Karmin et al., 2015), becoming universal
in non-Africans.
Evolutionary gap: bottleneck looks random under Darwinian models.
APH view: selective reset — a genetic “switch” coinciding with Adamic
ignition.
14.2.3 The Pattern
Individually, each locus could be explained as evolutionary noise. But
collectively, the pattern is undeniable:
Installed capacities (FOXP2, HAR1, SRGAP2C, ARHGAP11B) long
before threshold.
Dormancy across 200,000+ years — statistically implausible.
Sudden synchronized ignition after bottleneck.
This is not convergence. This is engineering.
