Page 21 - Aloe Vera Information - Scientific Papers about Aloe Vera

Yeasts and trophozoites, contrary to what was previously believed, do not appear to be normal
inhabitants of the mouth. They are associated with oral and/or systemic disease, and if left untreated in an
apparently symptomless host, will lead to the deterioration of the oral and general health of the patient
(Lyons, p 73, p. 15).

Dr. Lyons, through meticulous and step-by-step development of his theory, proves that these heretofore
accepted, “normal” inhabitants should be our target organisms in the treatment of many oral and systemic
diseases. He has documented and published proof that the elimination of those parasites restores the host
to a state of well being.

Another forward-thinking holistic dentist, Dr. Douglas Cook, from Surfing, WI, read between the lines
of Dr. Lyons’ work and gleaned the idea of why the Aloe Vera is so effective on so many people. In an
interview with Dr. Lyons, he confirmed this information. Aloe Vera is one of the most potent
protozoaand yeast-killing solutions that he had ever worked with. However, Dr. Lyons did not have at
his disposal, nor did he know about, these new, highly concentrated Whole Leaf Aloe Vera solutions.

Most of Dr. Lyons’ successes, which are monumental, were produced using traditional allopathic
medicine. Many of these medicaments used are highly toxic to the host, as well as the target organisms.
Possibly, nature has provided the perfect solution to this parasite problem: Aloe Vera.

In 1929, Kofoid reported finding Entamoeba in the bone marrow of some arthritic subjects. In 1981,
Snyderman and McCarty reported similar pathology in rheumatoid arthritis and destructive periodontal
disease. In 1982, Dr. Paul Keys, the former head of dental research at the United States National Institute
of Health, reported the almost invariable relationship between oral protozoa and periodontal deterioration.
E. gingivalis found at the base of periodontal pockets and E. histolytica found in ulcers of the colon
(Lyons, p. 28) behave similarly, causing the lesion to spread laterally as the amoebae migrate parallel to
the floor of the ulcer.

Could these one-celled animals really be that destructive? R. Mueller, in 1988, reported a new theory of
enzyme destruction. Polymorphs produce a proteolytic (protein-digesting) enzyme, “elastase” which is
normally bound to a circulating liver enzyme, “proteinase inhibitor” forming “elastase proteinase
inhibitor complex” (EPIC). The leukocytes are disrupted by the contact with amoebae leaving the
leukocytes in an uncontrolled state of maximum production and release of elastase. The EPIC balance
becomes overpowered, leading to rapid, uncontrolled, lytic activity. This concept fits well with the
understanding that destructive periodontal disease may be considered an autoimmune disease (Genco and
Mergenhagen, 1982) and shows that “the supreme irony of this state of parasitism is that the very cells
which should protect the host in fact destroy the host and are then, in turn, consumed by this predator
parasite, Entamoeba gingivalis.” (Lyons, p. 34)

These parasites also are capable of being infected with a virus. From within this safe harbor protected
from the patient’s immune response, a continuous stream of pathogenic particles could eventually destroy
the host. Could this be the reason why Epstein-Barr and HIV patients are helped with Aloe Vera? Does
the Aloe Vera destroy the virus’ hideout? Whatever research eventually shows, the destruction of these
one-celled invaders will leave the host with a stronger immune system.

Many dentists have been dismayed over the rampant decay in the cervical areas of some of their
patients’ teeth. These lesions tend to rapidly spread along and below the gingival margin. Within just a
few months this mostly painless destruction may render the tooth unsalvageable. Dr. Lyons has shown
that this rapid decay is caused by Candida, which is capable of both aerobic and anaerobic metabolism.

Antibiotics and antifungals have proven effective if the right ones at the right times are used. Reports
from the literature show that neither E. gingivalis nor E. histolytica is capable of initiating infection