Page 363 - Equine Clinical Medicine, Surgery and Reproduction, 2nd Edition
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338                                        CHAPTER 1



  VetBooks.ir  1.663                                      1.664




















                                                          Fig. 1.664  Transverse scan obtained from the
                                                          palmarolateral aspect of the pastern, immediately
                                                          distal to the base of the lateral PSB in a horse with
                                                          severe injury to the DSLs. The ODSL is enlarged
                                                          with loss of definition of its contours (yellow arrows).
           Fig. 1.663  Similar view as in 1.657. This horse   There is heterogeneous decrease in echogenicity.
           sustained partial breakdown of the distal sesamoidean   A small, avulsed bone fragment is visible (red arrow).
           ligaments (DSLs). The short (deep) DSLs are ruptured
           with frayed proximal fragments visible (yellow   roughening and irregularity of the PSB surface at
           arrows). The straight DSL was partially torn from the   the ligament origin (Fig. 1.664). Chronic injuries
           proximal scutum, with amorphous tissue filling the   are more commonly encountered, with loss of nor-
           space between the scutum and ligament (red arrow).   mal  fibre  pattern  and  mineralisation (Fig. 1.665).
           This was associated with fragmentation of the base of   SDSL injuries are most often localised in the middle
           the PSB (not visible in this image).           portion of the ligament. A discrete, hypo- to anecho-
                                                          genic core lesion is visualised, although diffuse
           Ultrasonography                                lesions are also encountered (Figs. 1.666, 1.667).
           This is a difficult anatomical area to scan and may   Rupture of the SDSL is uncommon but will be evi-
           require some experience. Each ligament must be   dent with complete loss of the ligament continuity,
           approached specifically from the base of the PSBs.   absence of fibre pattern and presence of a poorly
           The  use of a  microconvex  array  transducer  may   delineated,  hypoechogenic  and  heterogeneous  area
           be helpful to evaluate the short and cruciate DSLs   separating the frayed proximal and distal portions
           (Fig. 1.657).  The  affected ligaments  are mark-  of the ligament (Fig.  1.668). Chronic lesions are
           edly enlarged, they appear hypoechogenic and het-  characterised by a marked increase in CSA, loss of
           erogeneous, and there is usually evidence of bone   normal fibre alignment, abnormal heterogeneity and
           remodelling at the sesamoidean and phalangeal   hyperechogenic foci. Focal, ectopic mineralisation
           insertion areas in chronic cases. Complete rupture   may be present. This may occasionally be an inci-
           is rare and is usually associated with either com-  dental finding but probably represents an old lesion
           plete DSL breakdown or fracture of the base of   that was overlooked.
           the PSBs (Fig. 1.663). ODSL desmitis presents as
           obvious thickening and heterogeneous decrease in  Magnetic resonance imaging and computed
           echogenicity of the affected ligament(s), most often  tomography
           near the sesamoidean origin. They can affect one or   MRI and CT may be useful to identify complex DSL
           both ODSLs and are rarely bilateral. Lesions may   injuries, and are most useful to look for proximal
           be diffuse or discrete and there is usually marked   DSL injuries.
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