Page 825 - Equine Clinical Medicine, Surgery and Reproduction, 2nd Edition
P. 825

800                                        CHAPTER 4



  VetBooks.ir  4.124                                      4.125




















                                                          Fig. 4.125  Horse with acute grass sickness showing
                                                          bilateral upper eyelid ptosis. (Photo courtesy
                                                          Graham Munroe)



                                                          paraphimosis in entire males and chronic rhinitis
                                                          sicca may be noted.

                                                          Differential diagnosis
           Fig. 4.124  Horse with acute grass sickness showing   A variety of other causes of acute colic, chronic colic,
           ptyalism. (Photo courtesy Graham Munroe)       chronic weight loss and dysphagia should be consid-
                                                          ered, but the most important factor is  differentiation
                                                          of EGS from causes of colic that would require
                                                           surgical intervention.
           than expected considering the apparent degree of
           abdominal pain. Body temperature is normal to  Diagnosis
           slightly elevated. Pytalism is common, as is dehydra-  Palpation p/r is important to identify other causes
           tion, which may be severe (Fig. 4.124). NG reflux   of colic. Dry, mucus-covered faeces are common.
           may be present and stomach rupture, with ensuing   Concurrent distended loops of small intestine recog-
           peritonitis, may occur if gastric decompression is not   nised on rectal palpation or transabdominal ultraso-
           performed. Dysphagia can be present but may not   nography, and voluminous foul-smelling gastric reflux
           be recognised because of the anorexia accompanying   may be present with the acute form and reflect gen-
           severe disease. Bilateral ptosis is invariably present in   eralised GI ileus. Large-colon and caecal impactions
           all forms of the disease (Fig. 4.125).         in the chronic cases reflect large intestinal ileus and
             The subacute form is similar to the acute form,   desiccation of the fibrous ingesta (Fig. 4.126). Clinical
           although of  lesser  severity.  Tachycardia  is usually   signs, signalment, identification of risk factors such as
           present; however, signs of distress or severe abdomi-  a recent change in grazing or previous disease on the
           nal pain are uncommon. NG reflux is uncommon.   premises and exclusion of other differential diagnoses
           Dry faeces are commonly present.               is suggestive of EGS. Temporary reversal of ptosis fol-
             The chronic form may be of insidious onset,   lowing the topical application of a 0.5% phenylephrine
           characterised by weight loss, depression, a ‘tucked-  ophthalmic solution to the conjunctival sac confirms
           up’ or ‘wasp-like’ abdomen, weakness and dyspha-  neurogenic smooth muscle paralysis as the cause of
           gia. Mastication is typically slow and laboured and   the ptosis. Barium swallow studies or oesophagoscopy
           oesophageal spasm may be noted after swallowing.   may confirm the retrograde flow of gastric fluid and
           Intermittent diarrhoea, bilateral nasal discharge,   abnormal oesophageal motility.
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