Page 1037 - Adams and Stashak's Lameness in Horses, 7th Edition
P. 1037

Occupational‐Related Lameness Conditions  1003


             reduced biochemical evidence of muscle damage in    Suspensory Desmitis
             Thoroughbreds with recurrent exertional rhabdomyoly­  Forelimb suspensory desmitis (SD) is common in
  VetBooks.ir  of rhabdomyolysis in racing Thoroughbreds.  Although   endurance horses, affecting both the proximal area and
             sis.  In addition, dantrolene reduced recurrent episodes
                3
                                                    1
                                                                 branches. Hindlimb SD is less common but likely has a
             dantrolene use during competition is prohibited, it is
             used in an attempt to prevent recurrent myopathy    similar pathogenesis, and the treatment options are
             during training and/or long‐distance transport of elite   identical. SD usually develops when local footing con­
             Arabian endurance horses predisposed to myopathy    sists of deep sand, soft soil, or mud. Hence, in areas such
             prior to resuming work.                             the United Arab Emirates, SD is the most common cause
               Muscle spasms or cramps and muscle strains are also   of lameness in endurance horses. The diagnosis is sus­
             quite common in the sport. The gluteal muscles are com­  pected based on palpation of the limb and is confirmed
             monly affected, but other muscle groups, including the   by ultrasonography (Figure 9.45). The author’s impres­
             gracilis, lumbar, triceps, biceps, forearm, and pectoral   sion is that medial/lateral hoof imbalance predisposes
             muscles, may be involved. Occasionally horses with   endurance horses to suspensory branch desmitis. With
             muscle spasms are acutely painful; they may collapse   branch lesions, usually only one branch is affected;
             and show signs similar to those of acute colic. These   therefore, notable swelling may be evident on palpation
             individuals usually benefit from prompt analgesic treat­  compared with the opposite branch or leg. In contrast,
             ment. Most often an α‐2 agonist (e.g. detomidine, xyla­  lesions of the proximal suspensory and body may be less
             zine) with or without a narcotic (butorphanol) is more   obviously swollen, and diagnosis is usually based on
             effective and safer compared with high doses of NSAIDs,   sensitivity to palpation and ultrasound. Subtle, recurrent
             considering the likelihood of significant concurrent   lameness  associated  with proximal  SD  may  require
             dehydration. Massage therapy, stretching or walking,   diagnostic nerve blocks followed by ultrasonography
             and topical heat may help treat muscle cramps. On‐site   for confirmation.
             serum biochemical analysis in these patients is prudent   After an acute injury, the character of the ultra­
             because it may reveal electrolyte imbalances, necessitat­  sonographic lesion severity may worsen over time, so
             ing correction.                                     serial examinations to follow lesion progression are
               Muscle pain affecting the back and skin sensitivity of   recommended. Response to topical ice therapy can be
             the saddle and girth regions is common in endurance   dramatic and is warranted for the first 24 hours’ post‐
             horses. It is likely that both are related to rider imbal­  injury.  This should be followed by intermittent ice
             ance and poor saddle fit and compensatory back pain   water or cold‐water therapy and bandage support. After
             may develop secondary to hock lameness. In the neck   rehydration, NSAID treatment for the first 5–10‐day
             region, sensitivity of the strap muscles in front of the   post‐injury is typical. The prognosis for return to func­
             shoulder (brachiocephalicus m.) is very common and   tion is good in horses with mild desmitis. Those with
             likely develops as a consequence of forelimb lameness   minimal to no ultrasonographic changes may only
             (usually in the feet or ankles) or fatigue.         need 3–4 weeks of stall rest and hand walking before
               More rarely, frank muscle tears may cause acute   gradually returning  to work under saddle, with an
             lameness. Occasionally, local hematomas may form.   additional 4–6 weeks of walk/light trot intervals prior
             Rest and NSAIDs (post‐rehydration) usually lead to full   to returning to training work. With significant lesion
             recovery.                                           size, long‐term rest (minimum 6–8 months) is prudent.
                                                                 Horses  with  significant  desmitis  are best managed





















             Figure 9.45.  Ultrasonographic image
             of the lateral suspensory branch from an
             endurance horse that developed grade 3
             lameness after 83 miles of competition.
             Enlargement and fiber disruption were
             minimal, and the horse returned to work
             after several weeks of rest. Source:
             Courtesy of Dr. Patty Doyle.
   1032   1033   1034   1035   1036   1037   1038   1039   1040   1041   1042