Page 237 - Adams and Stashak's Lameness in Horses, 7th Edition
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Diagnostic Imaging   203




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                                                                 Figure 3.19.  Lateromedial (LM) projection of the metacar­
                                                                 pophalangeal joint with marked supracondylar bone loss on the
             Figure 3.18.  Dorsolateral to palmaromedial oblique (DLPMO)   palmar cortex of distal MCIII (short arrow), secondary to chronic
             projection of the metacarpus showing a well‐defined osseous body   inflammation and effusion within the joint.
             (sequestrum) surrounded by a radiolucent rim (cloaca) on the mid‐
             diaphysis of MCIV. There is mild surrounding sclerosis and a small
             periosteal reaction just proximal to the cloaca, representing the
             involucrum (arrow).


             Trabecular Bone
               Generalized bone opacity may be decreased or
             increased. Decreased bone density is seen secondary to
             chronic moderate to severe lameness resulting in decreased
             load bearing. The osteopenia that develops in these limbs
             can be recognized radiographically as a coarse trabecular
             pattern with or without thin cortices (Figure 3.22).
               Increased bone opacity, often referred to as sclerosis,
             is identified radiographically with loss of trabecular pat­  Figure 3.20.  Palmaroproximal to palmarodistal oblique
             tern secondary to bone deposition within the medullary   (Pa45Pr‐PaDiO, skyline) projection of the navicular bone. On the
             cavity (Figure  3.23). Sclerosis is a common adaptive   flexor aspect of the navicular bone, note the poor corticomedullary
                                                                 definition, irregular medullary sclerosis, and cortical erosions
             response to load bearing and is frequently found in the   consistent with advanced degenerative changes.
             third carpal bone and central and third tarsal bones
             (Figure 3.24). Increased bone density is also routinely
             seen at proximal MCIII or MTIII at the origin of the   route of infection. Both acute and chronic osteomyelitis
             suspensory ligament. 23,56                          can be identified radiographically (Figures  3.25 and
                                                                 3.26). It generally takes 7–10 days after clinical signs of
             Radiographic Signs (Bone Response Patterns)         acute osteomyelitis are observed before the earliest
             with Osteomyelitis                                  detectable radiographic bone changes occur because at
                                                                 least 50% of mineral content must be depleted from the
               Osteomyelitis in equine limbs may be hematogenous   bone to be radiographically visible.
             in  origin  or  result  from  penetrating  wounds  or  open   Because osteomyelitis can affect any bone in an equine
             fractures. The region affected depends on the source and   limb and must be differentiated from other focal bone
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