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420  Section 5  Critical Care Medicine

            pacing is begun. Once pacing has begun, the voltage of   bypass, significantly limiting its availability in veterinary
  VetBooks.ir  the pacing leads is gradually increased until they are   medicine at this time.
            “captured” by the heart. Evaluation of ECG, pulse oxime-
            try or digital palpation of the pulses can be used to verify
            capture. Transcutaneous pacing is always a stopgap     Prognosis
            measure to allow time for the placement of either a tem-
            porary transvenous pacing mechanism or a permanent   Published mortality rates for cardiogenic shock in veteri-
            pacemaker and should not be considered if these options   nary patients are lacking, but many human studies report
            are not available.                                mortality rates of 50–80%. In most instances, the devel-
             Management of caval syndrome secondary to heart-  opment  of  cardiogenic  shock  is  a  sequela  to  a  severe
            worm infection often requires direct removal of the   underlying structural or progressive conductive disease.
            worms from the right heart. The use of a transvenous   Therefore, both the short‐ and long‐term prognosis
            approach has been well described and, provided the dog   should  be  considered  guarded  to  poor.  Patients  that
            survives  the  perioperative  period,  appears  to  be  very   recover from cardiogenic shock are usually left with resid-
            effective for management of acute caval syndrome.   ual cardiac dysfunction that is irreversible in nature. The
            Surgical management is also the best treatment for well‐  exception to this may be dogs that are treated for caval
            defined intracardiac tumors. Unfortunately, open‐heart   syndrome secondary to canine heartworm disease and
            surgery requires total venous inflow occlusion or cardiac   animals receiving a pacemaker for bradyarrhythmias.



              Further Reading

            Bonagura JD, Schober KE. Can ventricular function be   outcome in 146 cats. J Vet Intern Med 2007; 21:
              assessed by echocardiography in chronic canine mitral   1002–7.
              valve disease? J Small Anim Pract 2009; 50(Suppl. 1):   Humm KR, Keenaghan‐Clark EA, Boag AK. Adverse
              12–24.                                            events associated with pericardiocentesis in dogs: 85
            Bove CM, Gordon SG, Saunders AB, et al. Outcome of   cases (1999–2006). J Vet Emerg Crit Care 2009; 19(4):
              minimally invasive surgical treatment of heartworm   352–6.
              caval syndrome in dogs: 42 cases (1999–2007). J Am   Pedro B, Lopez‐Alvarez J, Fonfara S, et al. Retrospective
              Vet Med Assoc 2010; 236(2): 187–92.               evaluation of the use of amiodarone in dogs with
            Brown AJ, Davison E, Sleeper MM. Clinical efficacy of   arrhythmias from 2003 to 2010. J Small Anim Pract
              sildenafil in treatment of pulmonary arterial hypertension   2012; 53: 19–26.
              in dogs. J Vet Intern Med 2010; 24: 850–4.      Yoon WK, Choi R, Lee SG, et al. Comparison of 2 retrieval
            Hall DJ, Shofer J, Meier CK, et al. Pericardial effusion in   devices for heartworm removal in 52 dogs with heavy
              cats: a retrospective study of clinical findings and   worm burden. J Vet Intern Med 2013; 27: 469–73.
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