Page 992 - Small Animal Clinical Nutrition 5th Edition
P. 992

Gastritis/Gastroduodenal Ulcers     1029



        VetBooks.ir  Box 52-1. Hairballs.


                    Hairballs occur commonly in cats because of their normal groom-
                    ing behavior and sharp barbs on the tongue that enhance hair  sance by many cat owners. Hairballs generally can be controlled.
                                                                     Various laxatives, lubricants, treats and foods are available for rou-
                    ingestion. Cats with longer, thicker coats and those with fastidious  tine management of these problems. Several commercial foods
                    grooming behavior usually have more problems with hairballs.  are available to help reduce the frequency with which cats vomit
                    Swallowed hair initially accumulates as loose aggregates or more  hairballs. Most of these foods have increased amounts of dietary
                    compacted, soft aggregates mixed with mucus. Hairballs are re-  fiber. Insoluble fiber, specifically cellulose, increases fecal hair con-
                    gurgitated periodically from the oropharynx or esophagus or vom-  tent as compared to other fibers when incorporated in complete
                    ited from the stomach, or they pass into the intestinal tract, where  foods. Kibble size is another important feature of foods designed
                    they are voided in the feces. Owners observe periodic gagging,  to reduce vomiting associated with hairballs. Radiographic gas-
                    retching and regurgitation or vomiting of hair and mucus (usually  trointestinal transit studies indicate that a larger kibble size is
                    not containing food or bile). Hairballs are often tubular.  associated with an increased tendency for hairballs to exit the
                      Trichobezoars are harder concretions within the stomach or  stomach and be eliminated in the feces, thereby reducing the fre-
                    intestines formed of hair, mucus and other material. Trichobezoars  quency of vomiting.There is little or no evidence to support the use
                    probably begin as simple aggregates of hair, but progress to larg-  of lubricants (e.g., petroleum jelly) or papain for the treatment of
                    er and harder concretions. They are less common in cats than typ-  hairballs in cats. If used, laxatives and lubricants should be given
                    ical hairballs, but are more likely to cause severe clinical signs.  intermittently because large daily doses may interfere with normal
                    Trichobezoars are a common cause of anorexia in pet rabbits  digestion and nutrient absorption.
                    (Chapter 70). Large trichobezoars may obstruct pyloric outflow or  Frequent regurgitation or vomiting of hairballs (i.e., every day)
                    the intestines and must be removed by surgery or endoscopy.  with or without diarrhea, weight loss, anorexia or abdominal pain
                      How cats eliminate aggregates of hair is probably similar to how  usually indicates an underlying problem (e.g., gastric motility
                    they eliminate the pelts of small mammals that are ingested as  defect or lymphoplasmacytic enteritis). Cats with severe or fre-
                    part of a natural diet. Cats that hunt frequently may be seen vom-  quent clinical signs should be evaluated more extensively with
                    iting the pelts of voles, mice, small rabbits and other mammals.  diagnostics including hematology, serum biochemistry profiles,
                    This may be a protective mechanism for eliminating less digestible  radiography and upper gastrointestinal endoscopy.
                    portions of prey.
                      Although hairballs do not usually cause significant clinical dis-  The Bibliography for Box 52-1 can be found at
                    ease, their associated clinical signs are considered to be a nui-  www.markmorris.org.


                  Water                                               Hypokalemia in association with GI disease will be particular-
                  Water is the most important nutrient for patients with acute  ly profound if losses are not matched by sufficient intake of
                  vomiting because of the potential for life-threatening dehydra-  potassium.
                  tion due to excessive fluid loss and inability of the patient to  Electrolyte disorders should be corrected initially with
                  replace those losses. Patients with persistent nausea and vomit-  appropriate parenteral fluid and electrolyte therapy. Foods for
                  ing should be supported with subcutaneous or intravenous  patients with acute gastroenteritis should contain levels of
                  rather than oral fluids. Moderate to severe dehydration should  potassium, chloride and sodium above the minimum allow-
                  also be corrected with appropriate parenteral fluid therapy.  ances for normal dogs and cats. Recommended levels of these
                                                                      nutrients are 0.8 to 1.1% potassium (dry matter [DM]), 0.5 to
                  Electrolytes                                        1.3% DM chloride and 0.3 to 0.5% DM sodium.
                  Gastric and intestinal secretions differ from extracellular fluids
                  in electrolyte composition, so their loss can result in systemic  Protein
                  electrolyte abnormalities. Dogs and cats with vomiting and  Foods for patients with acute gastritis and/or gastroduodenal
                  diarrhea may have low, normal or high serum potassium, chlo-  ulcers should probably not provide excess protein (no more
                  ride and sodium concentrations. The derangement that pre-  than 30% for dogs and 40% for cats). Products of protein diges-
                  dominates in a particular animal depends on several factors,  tion (peptides, amino acids and amines) increase gastrin and
                  such as the severity of the disease, nutritional status of the  gastric acid secretion (Feldman and Grossman, 1980; Delvalle
                  patient and site of the disease process. Serum electrolyte con-  and Yamada, 1990).
                  centrations are helpful in tailoring appropriate fluid therapy  Some authors recommend “hypoallergenic” or elimination
                  and nutritional management of these patients. Mild hypo-  foods for patients with chronic idiopathic gastritis because
                  kalemia, hypochloremia and either hypernatremia or hypona-  dietary antigens are suspected to play a role in the etiopatho-
                  tremia are the electrolyte abnormalities most commonly associ-  genesis (Guilford, 1997). In some cases, elimination foods may
                  ated with acute vomiting (and diarrhea).            be used successfully without pharmacologic intervention be-
                    Total body depletion of potassium is a predictable conse-  cause mild to moderate chronic gastritis may respond to dietary
                  quence of severe or chronic GI disease because the potassium  management alone. Ideal elimination foods should: 1) avoid
                  concentration of gastric and intestinal secretions is high.  protein excess (16 to 26% for dogs; 30 to 40% for cats), 2) have
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