Page 387 - AWSAR 2.0

and gap junction intracellular communication channels were severely impaired in hepatocarcinogenesis and improved after mito-TEMPO treatment. Normalization of lipid peroxidation and increase of enzymatic and non-enzymatic antioxidant defence systems in mito-TEMPO treated tumours showed that this agent also helped cells in maintaining normal
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cytoplasmic oxidative status.
Isn’t this amazing work, friends! The
findingcouldeffectivelyprotectagainstNDEA- induced hepatocarcinogenesis by inhibiting mitochondrial ROS that, in turn, modulates the course of disease. I wish I had known this before, but do not worry the “liver is happy” to share this with you and your buddies.