Page 797 - The Veterinary Care of the Horse
P. 797

PERITONITIS



  VetBooks.ir  Peritonitis is inflammation of the peritoneum. The peritoneum is a single layer of cells that

        lines  the  abdominal  cavity  and  the  organs  contained  within  it.  It  overlies  a  thin  layer  of
        connective tissue with blood vessels, lymphatic vessels and nerves. The abdomen contains a

        small amount of peritoneal fluid which is continually produced and absorbed. It minimizes
        friction  between  the  abdominal  organs  which  is  essential  for  normal  functioning  of  the

        gastrointestinal tract and when the animal moves.



        Causes

        There  are  many  causes  of  peritonitis.  In  horses,  it  is  usually  widespread  throughout  the

        abdomen  and  secondary  to  some  other  condition.  The  peritoneum  can  become  inflamed
        following mechanical damage or exposure to toxins and bacteria. This occurs in a number of

        ways.
        •    As the result of penetrating external injuries, e.g. a stake wound.

        •    Following an episode of colic when the blood supply to the gut is reduced and bacteria

             and toxins leak into the abdomen.
        •    Following  perforation  of  a  gastric  ulcer  or  rupture  of  any  part  of  the  gut  allowing

             intestinal contents into the abdomen.
        •    Rupture  of  any  other  abdominal  or  pelvic  organ,  e.g.  the  spleen,  uterus  or  bladder,

             exposes the peritoneum to bacteria and chemical irritants such as urine.

        •    Rupture of abdominal abscesses, e.g. from Streptococcus equi (strangles) infection.
        •    Septicaemic foals may develop peritonitis.

        •    Castration complications.

        •    Following abdominal surgery.

        •    Through migration of intestinal parasites.



        WHAT ACTUALLY HAPPENS WITHIN THE ABDOMEN?

        Once the peritoneum becomes inflamed, the blood vessels dilate and more peritoneal fluid is
        produced. Neutrophils migrate into the abdomen to help the peritoneal macrophages clear up

        the bacteria. Fibrin clots are deposited on the surfaces of organs and fibrous adhesions begin
        to develop (Figure 17.19).
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