Page 768 - Equine Clinical Medicine, Surgery and Reproduction, 2nd Edition
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Gastrointestinal system: 4.1 The upper gastrointestinal tr act                  743



  VetBooks.ir  4.45                                      4.46


















          Fig. 4.45  This section of an extracted tooth shows
          four vital pulp horns with one single necrotic pulp
          (arrows).



          cheek teeth. Mandibular teeth 307, 308, 407 and
          408 are most frequently affected in the lower jaw
          and maxillary teeth 108, 109, 208 and 209 are most
          commonly involved in the upper jaw.
            The aetiology of dental apical infections is unclear.
          It is possible that the mechanisms for apical infection
          are slightly different in maxillary and mandibular
          teeth. It is probable that anachoretic (blood borne)
          inoculation of the pulp via the apical vasculature   Fig. 4.46  A unilateral maxillary swelling typical of
          is the most likely mechanism. It has also been sug-  apical pulpitis of the maxillary premolars (arrows).
          gested that infundibular cemental hypoplasia, which
          has been identified in ultrastructural examination of
          maxillary teeth, may predispose to apical infection,   rostral maxillary teeth are infected), sinusitis and
          although this does not appear to be the mechanism   discharging tracts (Fig. 4.46). It is probably painful
          in all cases. However, the mandibular teeth lack   initially, although many cases present chronically,
          infundibula and are recorded with apical infections   when the pulp innervation is necrosed and the con-
          with a similar frequency to maxillary teeth. Bacterial   dition apparently non-painful. Occlusal exploration
          contamination of the pulp, after penetration of the   with a probe will reveal dentinal occlusal fissures in
          porous dentine on the occlusal surface, or subse-  over 50% of cases and these should be considered
          quent to fissure fractures as occurs in humans, may   significant, especially in conjunction with diagnostic
          be an initiating event. It has also been suggested that   imaging changes (Fig. 4.47).
          congenital enamel defects predispose to structural
          weakness in the tooth, which when exposed to the  Clinical presentation
          forces of mastication result in fractures that create   Unilateral maxillary and mandibular swellings, with
          physical communication between the occlusal sur-  ipsilateral submandibular lymphadenopathy are
          faces and pulp cavities of a tooth, resulting in pulp   typical. Eventually, the apical infections can absces-
          infection. The pathological fracture of teeth along   sate and discharge through tracts in the thin ventral
          such fault lines is commonly observed in maxillary   mandible for affected lower teeth. Maxillary apical
          cheek teeth but rarely in mandibular teeth.    infections rarely discharge through the maxillary
            Pulpitis presents as clinically detectable unilat-  bone, except after aspiration or trephination over
          eral  swellings  on  the  mandible  and  maxilla  (when   the swelling is attempted. Caudal (Triadan 08–11)
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