Page 793 - Equine Clinical Medicine, Surgery and Reproduction, 2nd Edition
P. 793

768                                        CHAPTER 4



  VetBooks.ir  Dissection of the melanoma from the gland tissue is   also be associated secondarily with other diseases
           virtually impossible, and radiation therapy has been
                                                          including rabies, oesophageal obstruction, pharyn-
                                                          geal dysphagia and stomatitis. Where the ptyalism is
           used to achieve remission in a few cases.
                                                          caused by a fungal infection, the excess salivation is
           PTYALISM                                       induced by the toxin slaframine, which is a parasym-
                                                          pathomimetic. The toxin may also induce lacrima-
           Ptyalism or excessive salivation may occur in   tion, anorexia and diarrhoea. Investigation should
           response to oral pain, periodontal disease, mucosal   be  directed  at  identifying the  primary cause  and
           penetration by a foreign body and ingestion of the   this should include an oral examination, pharyngeal
           fungus Rhizoctonia legumincola. The clinical signs may   endoscopy and passage of a nasogastric tube.



           DISEASES OF THE OESOPHAGUS

           INTRODUCTION                                   long that prevents oesophageal reflux under normal
                                                          circumstances.
           Anatomy of the equine oesophagus
           The equine oesophagus consists of stratified squa-  Clinical signs of oesophageal disease
           mous mucosa within a spiralling muscular tube,   Oesophageal disease often presents as signs of swal-
           which contains both striated and smooth muscle   lowing difficulties. Dysphagia, hypersalivation,
           in the cranial two-thirds and smooth muscle layers   and sweating are all associated with oesophageal
           only in the caudal third. There is no serosal layer   obstruction (Fig. 4.95). Dysphagia can have oral,
           and the outer adventitial layer is suspended within   pharyngeal and oesophageal causes that can be due
           the mediastinium. The oesophagus occupies a posi-  to inflammation, neurological dysfunction, pha-
           tion dorsal and slightly to the left of the trachea   ryngeal or oesophageal trauma or luminal obstruc-
           and is closely associated with the carotid arteries   tion. Aspiration of food spilling from an obstructed
           within the carotid sheath, the cranial sympathetic   oesophagus will also result in coughing and can
           trunk and the recurrent laryngeal nerves. The   lead to aspiration pneumonia. Perforation of the
           cranial oesophageal sphincter is about 5 cm long   oesophagus can lead to a painful mediastinal cellu-
           and comprises the cricopharyngeus and thyropha-  litis, while oesophageal reflux syndrome in foals can
           ryngeus muscles, which occlude the oesophageal   cause intermittent signs of oesophageal pain associ-
           lumen when contracted. The cranial dilation of the   ated with food ingestion.
           oesophageal entrance is in close approximation to
           the muscular processes of the larynx that lie ven-  Diagnosis of oesophageal diseases
           tral to and just caudal to it. During swallowing, the  Physical examination
           food bolus is voluntarily passed into the pharynx   A physical examination should include careful pal-
           using the tongue and pharyngeal muscles under a   pation of the neck from the larynx to the thoracic
           complex mechanism involving cranial nerves IX,   inlet for signs of swelling, pain, oedema, cellulites,
           X, XI and XII. The larynx is moved ventrally and   crepitus or firm masses. General physical param-
           caudally, the epiglottis is retroverted as the bolus   eters such as circulatory function, hydration status,
           passes  into  the  oropharynx  and  the  oesophageal   haematological  parameters,  electrolyte  imbalances
           sphincter muscles relax, allowing the bolus to pass   and neurological examination of the cranial nerves
           into the oesophagus. Thereafter, swallowing is an   should also be included.
           involuntary reflex controlled by branches of  the   Cautious passage of a round-ended, well- lubricated
           vagus (X) nerve. The oesophagus enters the stom-  nasogastric (NG) tube should be attempted. It may
           ach via the cardiac or caudal oesophageal sphincter,   be advisable for the horse to be sedated for this pro-
           which is a tight muscular ring approximately 10 cm   cedure. The tube is advanced into the nasopharynx
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