Page 798 - Equine Clinical Medicine, Surgery and Reproduction, 2nd Edition
P. 798

Gastrointestinal system: 4.1 The upper gastrointestinal tr act                  773



  VetBooks.ir  Clinical presentation                     4.100
          The clinical signs are non-specific, including signs
          similar  to  oesophageal  obstruction.  Discomfort
          when ingesting including gagging, bruxism, hyper-
          salivation and anorexia may all be exhibited. The
          inflammation can also cause oesophageal hypomo-
          tility, with secondary obstruction with food if the
          horse continues to eat.

          Differential diagnosis
          Colic in foals, gastric ulceration, cardiac sphincter
          stenosis, oesophageal obstruction.

          Diagnosis
          Thorough oesophagoscopy after clearing obstruc-  Fig. 4.100  Ulcerated mucosa in a foal with reflex
          tions to examine for signs of oesophagitis is   oesophagitis.
            indicated (Fig. 4.100). The presence of patchy or
          linear ulceration is diagnostic for oesophagitis.
          A careful examination for any underlying cause   their fibrinolytic activity to help reduce the like-
          including gastroscopy is necessary to prevent   lihood of fibrous stricture formation at the distal
          chronic recurrence.                            oesophageal sphincter. Their use should be care-
                                                         fully restricted in neonates to avoid increasing the
          Management                                     possibility of gastric ulceration. Gastric protectants
          Any primary cause such as oesophageal obstruc-  such as sucralfate are of questionable benefit but
          tion should be removed. Treatment with H2      may contribute by giving physical protection to the
          receptor antagonists (e.g. ranitidine or cimeti-  ulcerated  mucosa.  The  feeding  of  semi-liquidised
          dine) or by using oral medication with omeprazole   food will help reduce the likelihood of oesophageal
          are   beneficial in reducing gastric acid production   obstruction at the distal oesophageal sphincter.
          and  ongoing  chemical  damage  of  the  oesopha-  Stricture of the distal oesophageal sphincter as a
          geal mucosa. Alteration in feeding regimes to   result of fibrosis is thought to be a consequence of
          promote continuous gastric outflow can also help   chronic reflux oesophagitis and is occasionally diag-
          reduce  symptoms.  NSAIDs  may  be  indicated  for   nosed in young and adult horses.


          ACQUIRED OESOPHAGEAL DISORDERS


          OESOPHAGEAL OBSTRUCTION                        of motility disorders within the oesophagus has been
          (‘CHOKE’)                                      cited as a cause but is unproven. Sugar beet pulp that
                                                         is inadequately soaked is the most commonly impli-
          Definition/overview                            cated foodstuff in the UK, although obstruction with
          Physical obstruction of the oesophagus with ingesta.  other foodstuffs can occur. Primary grass choke is
                                                         rare and its occurrence suggests an underlying prob-
          Aetiology/pathophysiology                      lem. Absorption of saliva by the food bolus results in
          Physical obstruction  of  the  oesophagus with  an   it swelling and the bolus can become lodged in the
          impacted food bolus is the most commonly diagnosed   oesophagus, commonly at the thoracic inlet where
          oesophageal disorder in the adult horse. Inadequate   the diameter is narrowest and where the oesophagus
          soaking of proprietary or preserved foods is cited as   changes direction. Spasm of the oesophageal muscle
          the most frequent predisposing cause. The presence   distal to the bolus can occur, preventing its further
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