Page 41 - The Miracle of the Blood and Heart

Blood: The Incomparable
Liquid of Life

toms of this "good" mutation include: acute attacks of abdominal
and joint pain, ulcers on the legs, defective red blood cells, and
severe anemia—often leading to death. One can only imagine what
the "bad" mutations are like! No wonder that H. J. Mueller, who
won the Nobel prize for his work on mutations, said: "It is entirely
in line with the accidental nature of mutations that extensive tests
have agreed in showing the vast majority of them detrimental to the
organism in its job of surviving and reproducing—good ones are so
rare we can consider them all bad." 21

Another point regarding this claim made by evolutionists
needs to be considered. The majority of carriers, who are not
themselves affected by the disease, live in Africa, where the
risk of malaria is very high. This allows any carrier of sickle cell
anemia, thanks to the so-called beneficial mutation, to pass the
defective gene on to his or her children. The spread of the gene
in this way increases the next generation's chances of receiving
a defective gene from both mother and father. Defective genes
being received from both parents means that the child will
inevitably suffer from sickle cell anemia. Or else if healthy
genes are inherited from both parents, that person will not be
a carrier and thus will have no immunity to malaria.
Indeed, sickle cell anemia, which develops with a change
in just one of the 287 amino acids in hemoglobin, leads to the
death of 25% of those who suffer from it. 22
Dr. Felix Konotey-Ahulu, a world famous authority on
sickle cell anemia and author of The Sickle Cell Disease Patient,
says these on the subject:
If you are resistant to malaria, you are more likely to survive to
pass on your genes. Nevertheless, it is a defect, not an
increase in complexity or an improvement in function
which is being selected for, and having more carriers in
Adnan
Oktar

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