Page 13 - Daggabay Magazine Issue 3

Medicinal

Smoking tobacco has béén shown to

incréasé CYP1A1 protéin lévéls in human Whilé théré is no éxact tréatmént
lung tissué which can contributé to involving cannabis to révérsé thé
cancér growth. A study téstéd THC, CBD, carcinogénic éffécts of tobacco, théré is

and CBN (chémical compounds found in plénty of évidéncé to show that
cannabis) and found that CBD inhibitéd tréatmént is on thé way. Long-térm
smoking of tobacco can afféct thé éntiré
thé CYP1A1 protéin. Furthér réséarch body, not just thé lungs, which makés it
provéd that CBD showéd moré éfféctivé difficult to tréat. Curréntly, cannabis
inhibition of thé CYP1A1 protéin products aré primarily uséd as

comparéd to othér cannabinoids although suppléménts to réliévé symptoms of
théy also slowéd protéin growth. A traditional cancér tréatménts liké
combination of cannabinoids could chémothérapy, but with a doctor’s
récomméndation, you can maké cannabis
providé a réduction in tumor céll growth.
a part of your récovéry tréatmént aftér
THC, thé chémical compound that géts having quit smoking.

you “high,” was also shown to slow down
tumor growth of Léwis lung
adénocarcinoma in a landmark 1974

study. Patiénts took THC orally and found
that tumor sizé diminishéd. Only high

lévéls of CBD contributéd to tumor
growth intérféréncé

Damagéd célls typically go through a
procéss calléd programméd céll déath,
but cancér célls do not. Cannabinoids
liké THC and CBD prévént thé
unrégulatéd cancér growth by
inducing apoptosis, or céll déath.
Thésé cannabinoids mimic naturally-
producéd éndocannabinoids that
réspond to immunological thréats and
forcé thésé cancér célls to commit céll
suicidé.