Page 208 - Feline Cardiology
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              Arrhythmias and Other Electrocardiographic


              Abnormalities








                Key Points


                •	Although	at	opposite	ends	of	the	heart	rate	spectrum,	tachycardias	and	bradycardias	may	produce	similar	overt	clinical	signs
                  (e.g.,	syncope);	however,	most	cardiac	arrhythmias	in	cats	produce	no	detectable	signs	and	instead	are	incidental	findings.
                •	The	electrocardiogram	(ECG,	EKG)	remains	the	diagnostic	test	of	choice	to	confirm	or	rule	out	cardiac	arrhythmias.	If	a	resting
                  ECG	is	insufficient,	portable	or	implantable	ECG	monitors	may	confirm	the	diagnosis.
                •	Myocardial	disease	is	often	present	in	cats	with	ventricular	tachyarrhythmias	(premature	ventricular	complexes,	ventricular
                  tachycardia)	or	atrial	fibrillation.






              INTRODUCTION                                       indistinguishable  in  their  clinical  manifestations  from
                                                                 those arrhythmias that cause an excessively slow heart
              Cardiac arrhythmias (dysrhythmias) are defined as dis-  rate  (bradycardias,  also  called  bradyarrhythmias).  The
              turbances in the rhythm of the heartbeat (Tilley 1993;   rapid fluctuations in heart rate brought on by sporadic
              Mirvis  and  Goldberger  2008;  Fox  and  Harpster  1999;   arrhythmias  can  alter  cardiac  output  faster  than  a
              Côté 2010). In cats, as in other species, a range of sever-  patient’s  homeostatic  mechanisms  can  offset  these
              ity exists in terms of the significance of an arrhythmia   changes. Therefore, forward cardiac failure, manifested
              to the patient: arrhythmias may be normal physiologic   classically as syncope, is the chief complaint of cardiac
              variants, such as sinus tachycardia in the exam room;   arrhythmias in cats, in contrast to chronic mechanical
              individual variants of no concern, such as right bundle   pump failure, such as with the cardiomyopathies or con-
              branch block; markers for underlying structural heart   genital  malformations,  where  backward  (congestive)
              disease, such as many cases of third-degree AV block or   heart failure occurs as a result of the lesion exceeding
              atrial fibrillation; or the principal cause of the patient’s   the body’s longstanding compensatory mechanisms.
              chief  complaint:  episodic  weakness,  syncope,  or  even   The rarity of syncope in the cat attests to the severity
              sudden death.                                      of  arrhythmia  needed  to  cause  an  observable  change.
                                                                 When bradycardias limit cardiac output, they do so by
              ETIOLOGY, PATHOPHYSIOLOGY, AND
              GROSS PATHOLOGY                                    compromising  heart  rate  (cardiac  output  [ml/min] =
                                                                 heart  rate  [beats/min] × stroke  volume  [ml/beat]).
              Broadly, arrhythmias can be divided into those disorders   When tachycardias limit cardiac output, they do so by
              where the creation or conduction of impulses is greater   limiting stroke volume: the physiologic maximum heart
              than  normal,  and  those  where  it  is  less  than  normal.   rate (the fastest rate of sinus tachycardia possible in a
              Both  situations  can  cause  identical  clinical  manifesta-  healthy cat, subjectively 260–280 beats/min for periods
              tions: arrhythmias that involve an excessively high heart   of  several  seconds  to  minutes)  ensures  adequate  dia-
              rate  (tachycardias,  also  called  tachyarrhythmias)  are   stolic  filling  time  for  the  ventricles,  but  pathologic



              Feline Cardiology, First Edition. Etienne Côté, Kristin A. MacDonald, Kathryn M. Meurs, Meg M. Sleeper.
              © 2011 John Wiley & Sons, Inc. Published 2011 by John Wiley & Sons, Inc.

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