Page 68 - Poultry-Punch April 2020 edition
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POULTRY PUNCH                                                                          ARTICLE

            Transmission:                                       In laying birds, ova can be congested and sometimes ruptured,
             The virus is transmitted via the respiratory secretions, as well as  with free yolk in the abdominal cavity. Desquamation of respiratory
            faecal droplets from infected poultry. Contaminated objects and  epithelium,  edema,  epithelial  hyperplasia,  mononuclear  cell
            utensils may aid transmission and spread of the virus from one  infiltration of the submucosa, and regeneration occur in various
            flock to another. Evidence of virus was shown in trachea, kidney,  combinations.  Repair  processes  begin  after  6–10  days,  and  are
            and Bursa of Fabricius 24 hrs following aerosol transmission. The  complete  in  14–21  days.  Some  virus  strains  affect  the  kidney,
            nature  of  IBV  persistence  remains  to  be  elucidated;  however,  causing interstitial nephritis.
            detection of the virus in the caecal tonsils (up to 14 weeks) and   Poor egg shall quality, rugged appearance, thin shells and
            from faeces (20 weeks) after infection might suggest a role of faecal  watery albumin. In hatching eggs curled and stunted embryos
            shedding in viral transmission and persistence.  are common

            Incubation Period of IB
             Generally  the  short  incubation  period  for  IBV  varies  with
            infective dose and route of infection. For example, while infection
            via the tracheal route may take a course as short as 18 hours, ocular
            inoculation leads to an incubation period of 36 hours


            Pathogenesis and Pathology
             The  virus  replicates  to  high  titer  first  in  the  respiratory  tract
            (ciliated epithelial cells); this is followed by viremia (within 1–2
            days of infection), which distributes the virus to many organs. The
            virus can cause extensive damage to the ovaries, oviduct, and the
            kidneys, but this is dependent on the properties of individual virus
            strains. The intestinal tract is another site of primary infection, but
            damage usually is minimal.
            The most frequent gross pathologic finding is mucosal thickening
            within  The  upper  and  lower  respiratory  tract,  with  serous  or
            catarrhal  exudate  in  the  nasal  passages,  trachea,  bronchi,  and
            airsacs. In very young chicks, the main bronchi may be blocked with
            caseous yellow casts. Pneumonia and conjunctivitis occur in some
            cases.



















                        Fig: a).Chick gasping for breath,
                    b).inflammation of larynx and  c) Nephritis
             Clinical Signs of Infectious Bronchitis
               l gasping
               l coughing
               l sneezing
               l tracheal rales
               l nasal discharge                               Shortening of oviduct in pullets causing “False Layers with normal
               l wet eyes                                    Ovary.Cystic oviduct distended with watery fluid




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