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AcknowledgementsThis study was supported partly by   glutathionedepletion with subsequent production of   cervical lesions. DentMater 2013;29:e271–80.[44] van
        the County of Västerbottenand Saremco AG.r e f e r e n   oxygen reactivespecies. J Biomed Mater Res A   Dijken JWV. A clincial evaluation of anteriorconventional,
        c e s[1] Davidson CL, Feilzer A. Polymerization   2003;66:476–82.[23] Spahl W, Budzikiewicz H,   microfiller and hybrid composite resin fillings:a six year
        shrinkage andpolymerization shrinkage stress in   Geurtsen W. Determination ofleachable components   follow up study. Acta Odontol Scand1986;44:357–67.
 950
        polymer-basedrestorations. J Dent 1997;25:435–40.[2]   from four commercial dentalcomposites by gas and   [45] Isokangas P, Alanen P, Tiekso J. The clinician´ıs
        Loguercio AD, Reis A, Ballester RY. Polymerization   liquid chromatography/massspectrometry. J Dent   ability toidentify caries risk subjects without saliva
        shrinkage:effects of constraint and filling technique in   1998;26:137–45.[24] Nakabayashi N, Watanabe A,   tests—a pilotstudy. Community Dent Oral Epidemiol
        compositerestorations. Dent Mater 2004;20:236–43.[3]   Gendusa NJ. Dentin adhesionof modified 4-META/  1993;21:8–10.[46] Seppä L, Hausen H, Pöllänen L,
 Table 6 – Published annual failure rates of the in Umeå and Copenhagen tested restorative systems in Class II restorations after 6 year follow up periods. AFR = annual  Davidson CL, de Gee AJ. Relaxation of   MMA-TBB resin: function of HEMA.Dent Mater   Helasharju K, Karkkainen S.Past caries recording made
 failure rate.  polymerizationcontraction stresses by flow in dental   1992;8:259–64.[25] Nakaoki Y, Nikaido T, Pereira PN,   in Public Dental Clinics aspredictors of caries
 Classification  Restorative system  Year of publica-tion  Failures at 6 years (%)  AFR (%)  Manufacturer  composites. J Dent Res1984;63:146–8.[4] Ferracane   Inokoshi S, Tagami J.Dimensional changes of   prevalence in early adolescence.Community Dent Oral
 (study follow up years)  (reference no)  JL, Mitchem JC. Relationship between   demineralized dentin treated withHEMA primers. Dent   Epidemiol 1989;17:277–81.[47] Siegel S.
        compositecontraction stress and leakage in Class V   Mater 2000;16:441–6.[26] Van Landuyt KL, Snauwaert   Nonparametric statistics. New York: McGraw-HillBook
 Resin composite system low  els/cmf  2016  11.4%  1.9  Saremco AG, Rebstein,
 shrinkage, HEMA/TEGDMA free  Switzerland  cavities. Am J Dent2003;16:239–43.[5] Lindberg A, van   J, De Munck J, Peumans M,Yoshida Y, Poitevin A, et al.   Company, Inc.; 1956. p. 166–72.[48] Stanislawski L,
 els/AdheSE (6 years)  20.0%  3.3%  Ivoclar/Vivadent, Schaan,  Dijken JWV, Hörstedt P. Interfacialadaptation of Class II   Systematic review of thechemical composition of   Daniau X, Lauti A, Goldberg M. Factorsresponsible for
 Liechtenstein  polyacid-modified resincomposites/resin composite   contemporary dental adhesives.Biomaterials   pulp cell cytotoxicity induced byresin-modified glass
 Resin composite, low  InTen-S/Excite  2015 [10]  12.8%  2.1%  Ivoclar/Vivadent, Schaan,  laminate restorations in vivo.Acta Odontol Scand   2007;28:3757–85.[27] Van Landuyt KL, De Munck J,   ionomer cements. J Biomed Mater Res1999;48:277–
 shrinkage  Liechtenstein  2000;58:77–84.[6] Bergenholtz G. Evidence for   Snauwaert J, Coutinho E,Poitevin A, Yoshida Y, et al.   88.[49] Mine A, De Munck J, Van Landuyt KL, Poitevin
 Resin composite,  Point 4/Optibond Solo Plus  14.3%  2.4%  Kerr Corp, Orange, USA
 microhybrid  (15 years)  dental  bacterial causation of adversepulpal responses in   Monomer-solvent phaseseparation in one-step self-etch   A, Kuboki T,Yoshida Y, et al. Bonding effectiveness and
 Resin composite, nanofilled  Ceram X/Xeno III  2015 [52]  10.1%  1.7%  DeTrey/Dentsply, Konstanz,  resin-based dental restorations. Crit RevOral Biol Med   adhesives. J Dent Res2005;84:183–8.[28] Ikeda T,   interfacialcharacterization of a TEGDMA/HEMA-free
 Ceram X/Excite (8 years)  5.8%  1.0%  Germany  2000;11:467–80.[7] Fleming GJP, Khan S, Afzai O,   DeMunck J, Shirai K, Hikita K, Inoue S, Sano H, et al.  three-step etch &rinse adhesive. J Dent 2008;36:767–
 Resin composite, nanofilled  Tetric Evo Ceram  2014 [53]  13.6%  2.3%  Ivoclar/Vivadent, Schaan,  Palin WM, Burke FJT.Investigation of polymerisation   Effect of air-drying on the strength of HEMA-rich   73.[50] Braga RR, Ballester RY, Ferracane JL. Factors
 highly filled hybrid  Tetric Ceram (10 years)  10.2%  1.7%  Liechtenstein  materials  shrinkage strain, associated cuspal movement and   versusHEMA-free one-step adhesives. Dent Mater   involved in thedevelopment of polymerization shrinkage
 Resin composite, hybrid  Spectrum TPH/Prime&Bond  2014 [55]  15,0%  2.5%  Dentsply DeTrey, Konstanz.  microleakage of MOD cavitiesrestored incrementally   2008;24:1316–23.[29] De Munck J, Ermis RB, Koshiro   stress inresin-composites: a systematic review. Dent
 (8 years)
 Resin composite, hybrid  Gradia Direct  2013 [56]  8.5%  1.4%  GC, Tokyo, Japan  3 3  with resin-based composite using anLED light curing   K, Inoue S, Van Landuyt K,Lambrechts P, et al. NaOCl   Mater2005;21:962–70.[51] Malhotra N, Kundabala M,
 Posterior/G-Bond  unit. J Dent 2006;35:97–103.[8] Versluis A, Tantbirojn   degradation of a HEMA-freeall-in-one adhesive bonded   Acharya S. Strategies to overcomepolymerization
 Resin composite, Giomer  Beautifil/FLbond (6 years)  17.7%  3.0  Shofu, Kyoto, Japan  ( 2 0 1 7 )  D, Douglas WH. Distribution oftransient properties   to enamel and dentin followingtwo air-blowing   shrinkage—Materials and techniques. Areview. Dent
 Resin composite, highly filled  Tetric Ceram/Excite  2011 [57]  14.0%  2.3%  Ivoclar/Vivadent, Schaan,  during polymerization of alight-initiated restorative   techniques. J Dent 2007;35:74–83.[30] Chang H-H,   Update 2010;37:2–10.[52] van Dijken JWV, Pallesen U.
 hybrid small-particle  Tetric Ceram/Tetric  12.3%  2.1%  Liechtenstein  composite. Dent Mater2004;20:543–53.[9] Versluis A,   Guo M-K, Kasten FH, Chang M-C, Huang G-F,Wang Y-L,   Eight-year randomized clinicalevaluation of Class II
 flow/Excite (7 years)  944–953  Tantbirojn D, Pintado MR, DeLong R, DouglasWH.   et al. Stimulation of glutathione 6 depletion,   nano-hybrid resin compositerestorations bonded with a
 Resin composite, fiber  Alert/Bond-1  2006 [58]  12.8%  2.1%  Jeneric/Pentron, Wallingford,  Residual shrinkage stress distributions in molars   ROSproduction and cell cycle arrest of dental pulp cells   one-step self-etch or a two-stepetch-and-rinse
 reinforced  CT, USA
 Nulite/NS Bond Universal  25.0%  4.2%  Nulite Systems International  aftercomposite restoration. Dent Mater 2004;20:554–  andgingival epithelial cells by HEMA.   adhesive. Clin Oral Investig 2015;19:1371–9.[53] van
 Adhesive (6 years)  PTY Ltd, Hornsby, Australia  64.[10] van Dijken JWV, Lindberg A. A 15-year   Biomaterials2005;26:745–53.[31] Paranjpe A,   Dijken JWV, Pallesen U. A randomized
 Ca-aluminate cement  Doxadent (3 years)  2005 [59]  21% after 3 years  7.0%  Doxa, Uppsala, Sweden  randomizedcontrolled study of a reduced shrinkage   Bordador LCF, Wang M-Y, Hume WR, Jewett A.Resin   10-yearprospective follow up of Class II nano-hybrid
 Resin composite (sandwich)  Z100/Vitremer (7 years)  2004 [60]  19%  3.2%  3M, St Paul, MN, USA  stress resincomposite in Class II cavities. Dent Mater   monomer 2-hydroxyethyl methacrylate (HEMA) is   andconventional hybrid resin composite restorations. J
 Resin composite smart  Ariston (3 years)  2002 [61]  26% after 3 years  8.7%  Ivoclar/Vivadent, Schaan,  2015;31:1150–8.[11] Burke FJT, Crisp RJ, James A,   apotent inducer of apoptotic cell death in human and   AdhesDent 2014;16:585–92.[54] van Dijken JWV.
 material  Liechtenstein
 Resin composite inlay  Brilliant/Brilliant duo  2000 [54]  11.5%  1.9%  Brilliant DI, Coltène AG,  Mackenzie L, Pal A, Sands P,et al. Two year clinical   mousecells. J Dent Res 2005;84:172–7.[32] Schweikl   Direct resin composite inlays/onlays: an11 year
 cement  Altstätten, Switzerland  evaluation of a low-shrink resincomposite material in UK   H, Schmalz G, Rackebrandt K. The mutagenicactivity of   follow-up. J Dent 2000;28:299–306.[55] Pallesen U,
 Resin composite  Fullfil/GC lining/enamel  14.7%  2.5%  DeTrey, Konstanz, Germany  general dental practices. DentMater 2011;27:622–30.  unpolymerized resin monomers in   van Dijken JWV, Hallonsten A-L, Halken J,Höigaard R. A
 bond (11 years)  [12] Magno MB, Nascimento GCR, da Rocha YSP,   Salmonellatyphimurium and V79 cells. Mutat. Res   prospective 8-year follow-up of posterior
        Ribeiro BPG,Loretto SC, Maia LC. Silorane-based   1998;415:119–30.[33] Schweikl H, Schmalz G, Spruss   resincomposite restorations in permanent teeth of
        composite resinrestorations are not better than   T. The induction ofmicronuclei in vitro by unpolymerized   children andadolescents in Public Dental Health Service:
        conventional composites—ameta-analysis of clinical   resin monomers. JDent Res 2001;80:1615–20.[34]   reasons forreplacement. Clin Oral Investig
        studies. J Adhes Dent2016;18:375–86.[13] Ferracane   Schweikl H, Spagnuolo G, Schmalz G. Genetic and   2014;18:819–27.[56] van Dijken JWV. A 6-year
        JL. Elution of leachable components fromcomposites. J   cellulartoxicology of dental resin monomers. J Dent   prospective evaluation of aone-step HEMA-free self
 composite systems containing low   systems with traditional monomers.   percentage of participants with   Oral Rehabil 1994;21:441–52.[14] Michelsen VB, Moe   Res2006;85:870–7.[35] Samuelsen JT, Hole JA, Becher   etching adhesive in Class IIrestorations. Dent Mater
 molecular weight monomers, is by   This shows that it is very well possible   parafunctional habits.But also by the   G, Ström MB, Lygre H. Quantitativeanalysis of TEGDMA   R, Karlsson S, Morisbak E,Dahl JE. HEMA reduces cell   2013;29:1116–22.[57] van Dijken JWV, Pallesen U.
 observing clinical studies with the   to exchange both HEMA and   inclusion of a high frequency extense   and HEMA eluted into saliva from twodental composites   proliferation and inducesapoptosis in vitro. Dent Mater   Clinical performance of a hybridresin composite with
 same study design and patient   TEGDMA from the resin composite   “amalgam cavities” in molar teeth,   by use of GC/MS and tailor-made internalstandards.   2008;24:134–40.[36] Kanerva, et al. Occupational   and without an intermediate layer offlowable resin
                                             allegic contact dermatitis from2-hydroxyethyl
        Dent Mater 2008;24:724–31.[15] Blomgren, et al.
                                                                                  composite: a 7-year evaluation. Dent
 selection. Table 6 presents posterior   system without changing the clinical   which increase the total fracture risk.   Adverse reactions in the oral mucosaassociated with   methacrylate and ethylene glycoldimethacrylate in a   Mater2011;27:150–6.[58] van Dijken JWV,
 resin composite studies performed by   durability. However,the good clinical   Wear was not observed to be a clinical   anterior composite restorations. J OralPathol Med   modified acrylic structural adhesive.Contact Dermatitis   Sunnegårdh-Grönberg K. Fiber-reinforcedpackable resin
 our research groups in Umeå and   results were only observed when els   problem, despite suggestions in the   1996;25:311–3.[16] Geurtsen W, Spahl W, Leyhausen   1995;33:84–9.[37] Kanerva L, Jolanki R, Estlander T.   composites in Class II cavities. J Dent2006;34:763–9.
                                                                                  [59] van Dijken JWV, Sunnegårdh-Grönberg K. A
                                             10 years of patch testingwith the (meth)acrylate series.
        G. Residualmonomer/additive release and variability in
 Copenhagen and published in   was combined with the cmf adhesive   literature that wear may be a   cytotoxicity oflight-curing glass-ionomer cements and   Contact Dermatitis1997;37:255–8.[38] Goon AT,   three-yearfollow-up of posterior cavities restored with
 international reviewer based dental   system. The resin composite els did   significant mode of failure in larger   compomers. J DentRes 1998;77:2012–9.[17] Geurtsen   Isaksson M, Zimerson E, Goh CL, Bruze M.   Doxadent. SwedDent J 2005;29:43–9.[60] Andersson-
 journals, of which the majority was   not shown optimal biomechanical   restorations, especially in patientswith   W, Spahl W, Muller K, Leyhausen G. Aqueousextracts   Contactallergy to (meth)acrylates in the dental series in   Wenckert I, van Dijken JWV, Kieri C. Thedurability of
        from dentin adhesives contain cytotoxic chemicals.J
                                                                                  extensive Class II open-sandwich restorationswith a
                                             southernSweden: simultaneous positive patch test
 41
 64
 published during the last 10 years   properties in earlier in vitro studies  ,   bruxing and clenching habits  . This   Biomed Mater Res 1999;48:772–7.[18] Geurtsen W,   reaction patternsand possible screening allergens.   resin-modified glass ionomer cement after six years.Am
 10,52–61 . Studies investigating Class   but combining the resin composite   confirms findings from recent reviews   Leyhausen G. Chemical–biological interactionof the   Contact Dermatitis2006;55:219–26.[39] Kanerva L,   J Dent 2004;17:43–50.[61] van Dijken JWV. Three-year
 IIrestorations-only, where their 6 year   with the HEMA/TEGDMA free   of clinical studies published during   resin monomer triethyleneglycoldimetacrylate(TEGDMA).   Alenko K, Estlander T, Jolanki R, Lahtinen A,Savela E.   performance of a calcium-,fluoride- and hydroxyl ions
                                                                                  releasing resin composite. ActaOdontol Scand
        J Dent Res 2001;80:2046–50.[19] Kaga M, Noda M,
                                             Statistics on occupational contact dermatitis from(meth)
 failure rates and annual failure rates   adhesive resulted in good durability.  thelast years  [62,63] . It can be   Ferracane JL, Nakamura W, Oguchi H, SanoH. The in   acrylates in dental personal. Contact   2002;60:155–9.[62] Ástvaldsdóttir A, Dagerhamn J,
 are shown. The results of two new   This adhesive has shown excellent   concluded that Class II resin   vitro cytotoxicity of eluates from dentin bondingresins   Dermatitis2000;42:175–6.[40] Kleverlaan CJ, Feilzer   van Dijken JWV,Naimi-Akbar A, Sandborgh-Englund G,
 restorative materials with early high   clinical results in Class VNCCL lesions   composite restorations performed with   and their effect on tyrosine phosphorylation of   AJ. Polymerization shrinkage andcontraction stress of   Tranæus S, et al.Longevity of posterior resin composite
        L929cells. Dent Mater 2001;17:333–9.[20] Goldberg
                                             dental resin composites. Dent Mater2005;21:1150–7.
                                                                                  restorations inadults. A systematic review. J Dent
 catastrophic failure rates are included,   41 .The main reason for failure was   the new TEGDMA/HEMA-free low   M. In vitro and in vivo studies on the toxicity ofdental   [41] Ilie N, Hickel R. Investigations on mechanical   2015;43:934–54.[63] Opdam NJM, van de Sande FH,
 indicating the need of shorter follow   material fracture followedby recurrent   shrinkage resin composite system   resin components: a review. Clin Oral   behavior ofdental composites. Clin Oral Investig   Bronkhorst E, Cenci MS,Bottenberg P, Pallesen U, et al.
 up times for new materials. The 88.6%   caries. This is in accordance with   showed good durability similar to   Investig2008;12:1–8.[21] Engelmann J, Leyhausen G,   2009;13:427–38.[42] Reichl F-X, Löhle J, Seiss M,   Longevity of posteriorcomposite restorations: a
                                             Furche S, Shehata MM, Hickel R,et al. Elution of
        Leibfritz D, Geurtsen W. Effectsof TEGDMA on the
                                                                                  systematic review andmeta-analysis. J Dent Res
 success rate observed after 6 years   recent findings in most of the clinical   hybrid or nanofiller resin composite   intracellular glutathione concentration ofhuman gingival   TEGDMA and HEMA from polymerizedresin-based   2014;93:943–9.[64] Ferracane JL. Is the wear of
 with the TEGDMA/HEMA-free resin   follow up  62,63 . The high relative   systems containing HEMA, TEGDMA   fibroblasts. J Biomed Mater Sci2002;63:746–51.[22]   bonding systems. Dent Mater 2012;28:1120–5.[43]   dental composites still a clinicalconcern? Is there still a
 composite system is in line with those   frequency fractures observed may be   or other low molecular weight   Stanislawski L, Lefeuvre M, Bourd K, Soheili-Majd   van Dijken JWV. A randomized controlled 5-year   need for in vitro wear simulatingdevice. Dent Mater
                                                                                  2006;22:689–92.
                                             prospectivestudy of two HEMAfree adhesives, a 1-step
        E,Goldberg M, Perianin A. TEGDMA-induced toxicity in
 of highly acceptable resin composite   explained partly by the high   monomers, with high effectiveness.  humanfibroblasts is associated with early and drastic   self etching and a3-step etch-and-rinse, in non-carious
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