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CHAPTER 66   Disorders of Peripheral Nerves and the Neuromuscular Junction   1161





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            FIG 66.6
            Contraction of the muscles on the left side of the face
            developed in an adult dog with a 2-month history of
            idiopathic left-sided facial nerve paralysis. Note the erect
            left ear and nasal deviation to the left.

            and cats with facial nerve paralysis caused by middle/inner
            ear disease also develop peripheral vestibular signs and/or
            Horner syndrome because of the close proximity of the
            nerves in the area of the middle and inner ear.
            Diagnosis
            Idiopathic facial nerve paralysis can be diagnosed only after
            excluding all other causes. A complete neurologic examina-
            tion should be performed to ensure there are no other cranial
            nerve deficits, ataxia, or proprioceptive deficits suggesting a
            brainstem lesion. Clinicopathologic testing (i.e., complete
            blood count [CBC], serum biochemistry profile, urinalysis)
            is required to evaluate for systemic or metabolic disease. A   B
            suspicion of hypothyroidism warrants evaluation of thyroid
            function (see Chapter 48).                           FIG 66.7
                                                                 (A and B) Skull radiographs of a 4-year-old Cocker
              All dogs and cats with facial nerve paralysis should be   Spaniel with bilateral otitis media resulting in bilateral facial
            evaluated carefully for disease of the middle and inner ear.   nerve paralysis. Both bullae chambers are opacified, and
            Careful otoscopic examination is important even if general   the left bulla is thickened by irregular and slightly indistinct
            anesthesia is required. Most animals with otitis media or   new bone.
            otitis interna have obvious otitis externa and a tympanic
            membrane that appears abnormal or ruptured, but occasion-
            ally the otoscopic examination is normal. If the suspicion for   facial nerve paralysis. If keratoconjunctivitis sicca is present,
            middle and inner ear disease is high, general anesthesia for   the eye should be medicated as needed. The paralysis may
            radiographs or CT and myringotomy to collect a sample   be permanent, or spontaneous recovery may occur in 2 to
            from the middle ear are warranted (Fig. 66.7).       6 weeks.

            Treatment                                            TRIGEMINAL NERVE PARALYSIS
            Medical treatment for bacterial otitis media/interna is dis-  Bilateral motor paralysis of the trigeminal nerves results in
            cussed in  Chapter 63. No treatment exists for idiopathic   sudden onset of an inability to close the jaw or prehend food.
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