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CHAPTER                               25
  VetBooks.ir

                Emergency Management


                    of Respiratory Distress













            GENERAL CONSIDERATIONS                               respiratory disease. Normal breath sounds may be increased
                                                                 in dogs and cats without respiratory disease, but crackles or
            Respiratory distress, or  dyspnea, refers to an abnormally   wheezes are not expected.
            increased effort in breathing. Some authors prefer to use   A physical examination should be performed rapidly,
            terms such as hyperpnea and increased respiratory effort in   with particular attention paid to the breathing pattern, aus-
            reference to this abnormality because dyspnea and distress   cultatory abnormalities of the thorax and trachea, pulses,
            imply feelings that cannot be determined with certainty in   and mucous membrane color and perfusion. Attempts at
            animals. Breathing difficulties are extremely stressful for   stabilizing the animal’s condition should be made before
            people and are likely so for dogs and cats as well. Dyspnea   further diagnostic testing is initiated.
            is also physically exhausting to the animal as a whole and to   Dogs and cats in shock should be treated appropriately
            the respiratory musculature specifically. Animals in respira-  (see  Chapter 28). Most animals in severe respiratory dis-
            tory  distress  at  rest  should  be  managed  aggressively,  and   tress benefit from decreased stress and activity, placement
            their clinical status should be frequently assessed.  in a cool environment, and oxygen supplementation. Cage
              A dog or cat in respiratory distress may show orthopnea,   rest is extremely important, and the least stressful method
            which is difficulty breathing in certain positions. Animals   of oxygen supplementation should be used initially. An
            with orthopnea will assume a sitting or standing position   oxygen  cage  achieves  both  of  these  goals,  with  the  dis-
            with their elbows abducted and neck extended. Movement   advantage that the animal is inaccessible. Sedation of the
            of the abdominal muscles that assist ventilation may be exag-  animal may be beneficial (Table 25.1). More specific therapy
            gerated. Cats normally have minimal visible respiratory   depends  on the location and cause of the respiratory dis-
            effort. Cats that show noticeable chest excursions or open-  tress (Table 25.2). Ventilatory support is needed for patients
            mouth breathing are severely compromised. Cyanosis, in   that are not adequately oxygenating despite appropriate
            which normally pink mucous membranes are bluish, is a sign   treatment or who have ventilatory failure. Oxygen supple-
            of severe hypoxemia and indicates that the increased respira-  mentation  and  ventilator  support  are  discussed  later  in
            tory effort is not sufficiently compensating for the degree of   this chapter.
            respiratory dysfunction. Pallor of the mucous membranes is
            a more common sign of acute hypoxemia resulting from
            respiratory disease than is cyanosis.                EMERGENCY MANAGEMENT BASED
              Respiratory distress caused by respiratory tract disease   ON LOCALIZATION
            most commonly develops as a result of large airway obstruc-
            tion, severe pulmonary parenchymal or vascular disease (i.e.,   LARGE AIRWAY DISEASE
            pulmonary thromboembolism), pleural effusion, or pneu-  Diseases of the large airways result in respiratory distress by
            mothorax. Respiratory distress can also occur as a result of   obstructing the flow of air into the lungs. For the purposes
            primary cardiac disease causing decreased perfusion, pul-  of these discussions, extrathoracic large airways (otherwise
            monary edema, or pleural effusion (see Chapter 1). In addi-  known as upper airways) include the pharynx, larynx, and
            tion, noncardiopulmonary causes of hyperpnea must be   trachea proximal to the thoracic inlet; intrathoracic large
            considered in animals with apparent distress, including   airways include the trachea distal to the thoracic inlet and
            severe anemia, hypovolemia, acidosis, hyperthermia, and   bronchi. Animals presenting in respiratory distress caused
            neurologic disease. Pain and steroid administration can   by large airway obstruction typically have a markedly
            cause tachypnea and should be considered among the dif-  increased respiratory effort with a minimally increased
            ferential diagnoses in patients without other evidence of   respiratory rate (see Table 25.2). Excursions of the chest may

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