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Musculoskeletal system: 1.5 The hindlimb                           183



  VetBooks.ir    1.5  The hindlimb



          Hindlimb lameness is common in the horse,      than the head nod typical of forelimb lameness.
          particularly in sport and general riding horses.   This chapter concentrates largely on conditions
          Riders often identify it as poor performance   affecting the hock and stifle as the distal
          rather than lameness, as a short stride and    limb, pelvis and soft-tissue lesions are covered
          increased pelvic excursion are harder to detect   elsewhere in other chapters.



          THE HOCK

          OSTEOARTHRITIS OF THE DISTAL                   may be a poor correlation between the severity of
          TARSAL JOINTS (BONE SPAVIN)                    lameness and the severity of radiographic changes.


          Definition/overview                            Clinical presentation
          Osteoarthritis (OA) of the tarsometatarsal (TMT)   The onset of lameness or poor performance is often
          and distal intertarsal (centrodistal) (DIT) joints   gradual. Horses may present with a variety of clinical
          and,  less  frequently,  the  proximal  intertarsal   signs ranging from a subtle change in performance
          ( talocalcaneal–centroquatral) (PIT) joint is the most   without obvious lameness, to moderate or severe lame-
          common cause of lameness involving the hindlimbs   ness. Occasionally, the first sign is identified by a farrier
          of mature horses. These are low-motion joints,   when the horse resists having the limb flexed during
          believed to act primarily as shock absorbers with   shoeing. Mild cases may warm out of the lameness and
          limited horizontal movement. Although it is usually
          seen in mature animals, bone spavin can occur in
          young Thoroughbreds, Warmbloods, Standardbreds   1.352
          and Western performance horses. ‘Juvenile spavin’
          is the term used for the condition in horses less than
          2 years old. The incidence of the condition appears
          to have little relationship to the type of horse or to
          the type or amount of work it has undertaken.

          Aetiology/pathophysiology
          Many factors may play a role in the development and
          progression of bone spavin. Normal ‘use trauma’ as
          well as abnormal conformation such as ‘cow’ or ‘sickle
          hock’ are believed to provoke formation of the con-
          dition. Excessive compression and rotational forces
          of the tarsal bones during jumps or stops and exces-
          sive tension of the ligaments over the dorsal aspect
          of the hock are all thought to be prominent in the
          development of bone spavin. Other factors include
          incomplete ossification and subsequent collapse of
          the third and central tarsal bones (Fig. 1.352), septic
          arthritis, fractures of the cuboidal tarsal bones and   Fig. 1.352  Lateromedial radiograph of the hock in a
          osteochondrosis (OCD). In Icelandic horses the dis-  young foal demonstrating collapse of the central and
          ease appears to have a hereditary component. There   third tarsal bones.
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