Page 101 - Clinical Pearls in Cardiology
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Auscultation of the Precordium 89


                   But this apparent fixed splitting usually disappears when
                   the individual assumes the sitting or standing position.
                   So the diagnosis of fixed splitting should be made only
                   after careful auscultation of S2 in the upright (i.e. sitting
                   or standing) position.
                7.  When do you say that P2 is loud?
                   The observation of an abnormally loud P2 component
                   of the second heart sound is almost always indicative
                   of pulmonary hypertension. P2 is normally inaudible
                   at the cardiac apex. Therefore, S2 appears to be single
                   at the apical area in normal persons (i.e. only the A2
                   component is audible). The occurrence of both the
                   audible components of S2 (i.e. A2 component and
                   P2 component) at the apical area is an indication
                   of increased intensity of the P2 component. In such
                   conditions, the P2 is said to be loud. (Ref: Synopsis of
                   cardiac physical diagnosis, Abrams. 2001).
                8.  What is the significance of S3?
                   The augmented early diastolic filling of the ventricle
                   (either due to increased left atrial pressure or due to
                   increased distensibility of a dilated ventricle) leads to a
                   vibratory motion of the ventricular walls, valve apparatus
                   and the inflowing blood mass. This vibratory motion
                   produces the low pitched S3 sound. It may be normally
                   heard in young individuals. However, an audible S3 in
                   an elderly person is always pathological.
                     The diastolic “pericardial knock” is a particular
                   form of S3 (loud and high-pitched) due to the sudden
                   distension of the left ventricle against the calcified
                   pericardium in constrictive pericarditis.
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