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14.9 Ununited Anconeal Process  209

             14.8   Osteochondrosis Dissecans

             Osteochondrosis/osteochondritis dissecans (OCD) of the elbow joint affects the medial humeral
             condyle (trochlea). In contrast to OCD lesions of other joints, elbow OCD is frequently diagnosed
             together with MCD. Similar to OCD lesions in other joints, OCD of the elbow is the consequence
             of an endochondral ossification failure resulting in excessive thickness of the cartilage that may
             detach (and develop a flap). Cartilage pathology affecting the trochlea may also develop secondary
             to a mismatch between the humerus and ulna, as well as from erosion from the opposing coronoid
             pathology. Such lesions are termed “kissing lesions” and can be difficult to distinguish from true
             OCD lesions in some cases (Cook and Cook 2009). Although OCD of the elbow can sometimes be
             identified with radiography, most often CT (Figure 14.5) or arthroscopy is used to establish a final
             diagnosis.  The  use  of  these  technologies  also  allows  to  identify  concurrent  incongruity  and
               coronoid pathology. Because the prognosis for elbow OCD is questionable, treatment is somewhat
             controversial, with most authors recommending surgical debridement of the lesion (and address-
             ing concurrent elbow pathology if indicated).


             14.9   Ununited Anconeal Process


             Failure of fusion of the ossification center of the anconeal process is defined as UAP. The patho-
             physiology of UAP is unclear, but premature distal ulnar physis closure frequently results in UAP   ELBOW REGION
             indicating that radioulnar incongruity may play a substantial role. Ossification of the anconeal
             process should be completed by 20 weeks during normal development, although fusion frequently
             occurs earlier. UAP has been reported to occur together with MCD in 16% of the cases (Meyer-
             Lindenberg et al. 2006). A wide variety of treatment options are available including ulnar osteot-
             omy procedures, lag screw fixation, or removal of the anconeal process.


             14.9.1  Signalment and History
             Any  large-/giant-breed  dog  is  susceptible  to  UAP,  especially  German  Shepherd  Dogs,  Bernese
             Mountain Dogs, and Mastiffs. Nevertheless, even small dog breeds, including French Bulldogs and
             Dachshunds,  have  been  diagnosed  with  UAP.  Animals  with  unilateral  disease  usually  present
             before they reach maturity. Bilateral disease, which is present in 20–25% of the cases, may make
             the lameness harder to identify for owners and hence these animals may present later in life due to
             advanced degenerative disease (Cross and Chambers 1997).


             14.9.2  Physical Exam
             Dogs with UAP generally have more obvious physical exam findings compared to dogs presenting
             with MCD. Significant joint effusion is generally palpable, and pain is most evident with hyperex-
             tension of the joint.


             14.9.3  Diagnostics
             In contrast to the other forms of ED, UAP is easily diagnosed with radiographs. If a lack of anco-
             neal fusion is observed after 20 weeks of age, the diagnosis is confirmed (Figure 14.8). The flexed
             lateral radiographic view (Figure 14.8A) eliminates superimposition of the anconeal process and
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