Page 240 - Canine Lameness
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212  14  Elbow Region

            been described to change pronation from approximately 30–60° after transection of the medial
            collateral ligament and supination from approximately 45–70° after transection of the lateral col-
            lateral ligament. However, a large inter-animal variation was also reported (Farrell et al. 2007).
            Therefore, comparison to the contralateral limb is recommended. NOTE: this test is also used to
            evaluate for a pain response in dogs with ED as discussed in Section 14.6.2.


            14.10.3  Diagnostics
            Elbow  luxations  (congenital  and  traumatic)  are  generally  suspected  based  on  history  (for
              traumatic  luxations)  and  palpation;  the  diagnosis  is  easily  confirmed  via  radiography.  For
            TEL, craniocaudal projections clearly show displacement of the radius/ulna while lateral pro -
            jections may be less obvious. The observer should also evaluate the radiographs for evidence
            of chronic changes (such as osteoarthritis) or avulsion fragments of the collateral ligament
            (Figure 14.9F) or other fractures since these findings may pose a contraindication for closed
            reduction and require surgical intervention. For CEL, radiographic changes vary based on the
            type of luxation and severity. If surgical treatment is planned, further imaging (CT) may be
            indicated to assess the integrity of the coronoid process and degree of elbow incongruity, and
            to aid in surgical planning.
       ELBOW REGION  14.11   Panosteitis






            Panosteitis is a developmental disease that affects the adipose components of the bone marrow of
            long bones. The disease has also been reported in the literature as eosinophilic panosteitis, juvenile
            osteomyelitis, as well as enostosis (i.e. medullary fibrosis). Histologically, the disease cycles through
            a phase where adipose bone marrow is replaced with osseous tissue followed by regeneration of
            the bone marrow to its original constitution. The disease was once thought to be of bacterial or
            viral origin; however, currently the etiology is controversial, and newer theories have included an
            association with high-protein diets and osseous compartment syndrome (Schawalder et al. 2002).
            Moreover, the high incidence in German Shepherd Dogs suggests a genetic component. The dis-
            ease is self-limiting and generally not associated with systemic signs. Treatment consists of rest
            and pain management.


            14.11.1  Signalment and History

            Dogs  affected  with  panosteitis  most  commonly  present  symptomatically  between  5  and
            18 months  of  age,  although  the  disease  has  been  reported  in  patients  up  to  5 years  of  age.
            Lameness can be of varying degrees of severity and may affect one or multiple limbs simultane -
            ously or sequentially since the disease frequently affects multiple bones (Bohning et al. 1970).
            The latter explains one of the hallmark signs of panosteitis, a lameness that shifts from one leg
            to another. Since the disease is self-limiting, owners may also report that the symptoms resolved
            without treatment. Recurrence may occur, yet in most cases subsequent episodes are less severe.
            Panosteitis is seen most frequently in German Shepherds (with a male predisposition), although
            many other large/giant breeds and even small-breed dogs are reported to be affected. As such,
            the disease should be considered a differential for any juvenile dog presented with shifting leg
            lameness (Towle-Millard and Breur 2018).
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