Page 78 - Canine Lameness
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50  4  The Neurologic Examination

            Table 4.2  (Continued)

                              Findings/observations  Interpretation
             Spinal reflexes  Normal                 Orthopedic or UMN; less commonly some LMN
                                                     (e.g. muscle or NMJ)
                              Increased/exaggerated  UMN, excitement, pseudohyperreflexia
                              Clonus                 UMN
                              Decreased              LMN, limited joint mobility, age‐related (patellar),
                                                     and excitement (tense; activation of extensor
                                                     muscles)
                              Absent                 LMN, limited joint mobility, age‐related (patellar),
                                                     and less commonly UMN (i.e. spinal shock or
                                                     myelomalacia)
                              Cutaneous trunci reflex  Cutoff: UMN, transverse thoracolumbar spinal
                                                     cord
                                                     Unilaterally absent: LMN (C8–T1 spinal cord
                                                     segments or lateral thoracic nerve), ipsilateral
                                                     Bilaterally absent: usually equivocal
                              Perineal               LMN (S1–S3 spinal cord segments or nerves)
             Perception of    Normal
             sensory stimuli and   Increased sensitivity   Orthopedic or neurologic
             pain             (hyperesthesia)
                              Reduced or absent sensitivity  Neurologic
                              (anesthesia)
                              Reduced nociception    Neurologic
                              (hypalgesia)
                              Absent nociception     Neurologic
                              (analgesia)

            CN, cranial nerves; GP, general proprioceptive; LMN, lower motor neuron; NMJ, neuromuscular junction; UMN,
            upper motor neuron.



            approaching every neurologic examination. Observation of a patient’s mentation, behavior, and
            posture should begin while taking a history and the dog is able to freely move about the room.
            Observations should continue throughout the examination to catch subtleties as the patient is
            moved from one position to another.
              Certain  components,  for  example  postural  reactions  (Box  4.3),  should  be  assessed  for  any
            patient as “screening” tests. For patients presenting with a lameness, Box 4.3 displays the mini-
            mal  components  of  the  neurologic  examination  that  should  be  performed.  However,  if  the
            patient or history is suggestive of neurologic dysfunction, a full neurologic examination should
            be completed.


            4.3.1  Mentation Status (Awareness) and Behavior
            Mentation is recorded in terms of level of consciousness as alert, dull, obtunded, stuporous, or coma-
            tose, and in terms of the content or quality of consciousness (e.g. inappropriate behavior). Since
            most neurologic conditions causing a monoparesis or lameness will involve the PNS, mentation
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