Page 995 - Veterinary Toxicology, Basic and Clinical Principles, 3rd Edition

Pieris (e.g., reactions, enteritis, and foot ragwort, other (Continued )

with Diseases include poisoning, jaundice allergic viral bacteremias, including disease sporidesmin eczema), nodularin or and compounds Ngaio

Association Other Differentials japonica of causes eczema), facial hypersensitivity or bacterial systemic viremias mouth and to Exposure (facial microcystis blooms), (algal hepatotoxic exacerbate may toxicity

watery and blistering mucous oral eyes, and appetite, of loss strong move; to include features and fatty liver; of cattle horses of salivation, excitement, and coma, onset ingestion; blind exhibit twitching, muscle excitement, convulsions, regurgitation; in amnesia of losses in herds beef high Island produced toxic secondary recovery very is constipation, dullness, pain, no occasionally

Toxidrome profuse Horses: diarrhea, of irritation membranes depression, pain, muscular disinclination necropsy ulceration gastric gastroenteritis, degeneration in toxidrome that resembles Picrotoxin-like: nausea, convulsions, sudden death; after h 24 48 may cattle charging, extreme epileptiform and bloating, produces stock humans; of 5% 10% South the honey country; is tutu from Hepatogenous photosensitization; survivors among severe slow; abdominal appetite, of loss rumination; icterus

a the to toxic in
more winter is Formerly primarily now often more are sheep poisoning in usually starved sheep; except surrounding the and is in (particularly horses attracted elephants poisoned individual leaves most losses pigs, most occur blow branches clippings out
Factors in feed of is it to or and occasionally are parts seeds toxin concentrated seeds); circus been of varies; serious sheep, horses; storms tree thrown

Risk Poisonings common when restricted. toxicity horses, reported cattle; in resistant Toxicity hungry cattle all poisonous petals the roots; leaves young not are tutu; have Toxicity plants normally part; cattle, and poisonings after down when or are

Distribution and islands; grasslands; shrubland; river to up terraces; of altitude m Throughout Zealand; in found areas in Common areas lowland far as Otago; as in common gardens

Livestock North south open along an 1500 New often stony coastal and forests south

to leaves from group); CNS, several Ngaione
Hazardous (dipterpene the in (lactone picrotoxin the in medullary poisoning and furanosesquiterpenoid oils. that protoxin hepatic activation

Are Toxins Prostratin acetate) bark and Tutin the localizes causing excitatory Ngaione other essential a is requires metabolic
That

Plants (mucous and on with excitation of glycine GABA A and

Zealand Diseases Irritant membranes skin); hypersensitivity; effect strong cardiovascular function Tetanus-like CNS (blocking spinal CNS and receptors) Hepatogenous damage photosensitization

New or

Major woody in cm branches dark oval, waxy red berries or narrow to flowers terminal usually laetum

of and Identification prostrate prostrate, 50 to scarred, black; to pointed greenish-gray, long; flowers, long, mm ovoid arborea opposed, from ovate to or fruit purple or
Summary Species Pimelea Small, branched shrub; diameter; are brown leaves mm B5 white 3 4 white Coriaria Leaves vary filiform lanceolate; axillary racemes; black Myoporum

63.3 of

TABLE VetBooks.ir Common Name Disease Strathmore Pimelea or New or Zealand Daphne poisoning Tutu toxicity Ngaio toxicity