Page 731 - Adams and Stashak's Lameness in Horses, 7th Edition
P. 731

Lameness of the Proximal Limb  697


             Acute Tenosynovitis of Extensor Tendons
               The long digital extensor tendon (LoDET) lies on the
  VetBooks.ir  cranial aspect of the crus and courses over the dorsum
             of the tarsus just lateral to the MTR of the talus. A prox-
             imal, middle, and distal extensor retinaculum cover the
             LoDET at the level of the tarsus. The tendon courses
             over the tarsus within a synovial sheath from approxi-
             mately the level of the LM and continues coverage
             through the distal retinaculum almost to the junction
             with the lateral digital extensor tendon (LDET). At this
             level, the conjoined tendons of the LoDET and LDET
             muscles continue distally similarly to the common digi-
             tal tendon of the front limb. The LDET of the hindlimb
             courses distally across the lateral surface of the tarsus.
               Direct trauma is the most likely cause of tenosynovi-
             tis of the extensor tendons of the tarsus. Blunt trauma to
             the dorsal and lateral surface of the tarsus can create
             acute tenosynovitis of extensor sheaths, particularly the
             LDET.  The LDET sometimes develops idiopathic or
             acute  tenosynovitis.  The  swelling within  the  synovial
             sheath may protrude proximally and distally to the reti-
             naculum of the lateral digital extensor. Lameness when
             it occurs is sudden in onset and moderate in severity.
             Mild distension of the sheath may occur initially. These   Figure 5.100.  Rupture of the peroneus tertius disrupts the
             effusions often persist in spite of appropriate treatment.   reciprocal apparatus allowing extension of the tarsus and fetlock
             The LoDET is rarely affected. The distal aspect of the PT   with the stifle flexed. Source: Courtesy of Dr. Gary Baxter.
             tendon can be injured as well. Diagnostic US should be
             utilized to assess the tendon for damage. Treatment is   Careful   monitoring of the healing process with US is
             determined based on the specific structure(s) involved. If   necessary to prevent reinjury.
             the tendon appears to be normal, then the treatment   Complete rest is the best treatment. The horse should
             may be directed at the synovial sheath.             be placed in a stall and kept quiet for at least 4–6 weeks,
                                                                 and then limited exercise should be given for the next
             Rupture/Tendonitis of the Peroneus Tertius          2 months. Most cases heal and show normal limb action,
                                                                 and if properly conditioned, most horses can return to
             (Fibularis Tertius)                                 normal work. After rupture of the PT tendon, 71% of
               The PT muscle is tendinous over its entire length. It   horses returned to their previous level of exercise with a
             arises from the extensor fossa of the lateral femoral con-  mean rehabilitation period of 41.5 weeks. If a horse was
             dyle and inserts with two distinct tendons to the tarsus.   a performance horse at the time of injury, it was 11 times
             Rupture of the PT is uncommon with trauma being the   less likely to return to its intended use. Furthermore, if an
             most common cause. 78,86,117,132  The muscle or tendon can   additional structure was injured at the time of rupture of
             rupture anywhere along its course and can result in an   the PT, the horse was 14.6 times less likely to return to its
                                                                            78
             avulsion fracture at its origin in the extensor fossa.   intended use.  Prognostic indicators previously reported
             When this muscle is ruptured, the stifle flexes, but the   for this type of injury include location and degree of rup-
             hock does not. Rupture of the PT is usually due to over-  ture. Prognosis has been reported to be favorable when
             extension of the hock joint (Figure 5.100). Entrapment   rupture occurs in the tibial region and poor if the rupture
             of a limb with violent struggles can cause this condition.   occurs at the point of origin from the extensor fossa of
             Rupture also may occur during the exertion of a fast   the lateral femoral condyle with any associated fracture.
             start causing overextension of the tarsus such as in   Performance horses have a decreased prognosis to return
             jumping. It can also occur after a full‐limb cast is applied   to their intended use after rupture of the PT.
             to the hindlimb. PT injury in adult horses may cause
             swelling of the distal cranial aspect of the crus near the
             TC joint (usually between the trochlear ridges), but   PERIARTICULAR TARSAL CELLULITIS
             lesions in the mid‐crus seem to be more common and
             often are not associated with significant swelling. If the   Spontaneous cellulitis can develop around the tarsus
             PT is ruptured, the tarsal joint can be extended while the   that can present as an unusual cause of an acute‐onset,
             stifle is flexed, indicating loss of the reciprocal appara-  severe non‐weight‐bearing lameness. One report
             tus function. There is often a dimpling that occurs within   described this condition in a series of TB racehorses.
                                                                                                               111
             the common calcaneal tendon. If the origin of the PT   Skin abrasions were identified in some horses some-
             fractures from the femur, femoropatellar effusion is a   where in the more distal part of the limb, and the onset
             prominent feature, and the gait deficit is similar. On US,   of swelling usually began on the dorsal aspect of the
             the acute ruptured PT is enlarged and shows multiple   hock. The cellulitis was quite painful to palpation. These
             small focal anechoic to hypoechoic lesions. On a longi-  clinical signs were associated with a fever, neutrophilia,
             tudinal scan,  the normal fiber orientation disappears.   and hyperfibrinogenemia. Prompt, aggressive treatment
   726   727   728   729   730   731   732   733   734   735   736