Page 147 - Equine Clinical Medicine, Surgery and Reproduction, 2nd Edition
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122                                        CHAPTER 1



  VetBooks.ir  Tendon damage is seen as focal signal increase   4 months. Horses should be shod with egg-bar shoes
                                                          or back-to-front shoes, thereby reducing the oppor-
           on both T1- and T2-weighted sequences, vari-
           ably accompanied by enlargement of the affected
                                                          with heel elevation may be useful in horses with
           lobe. There is a good correlation between the MRI   tunity for hyperextension of the DIP joint. Shoeing
           appearance and the pathological classification of   severe lameness to improve lameness quickly, but it
           lesions. Most tendon lesions are seen in the proximal   has been suggested this should not be maintained for
           recess of the navicular bursa. Lesions may be asso-  longer than 3 months to avoid permanent functional
           ciated with herniation of torn fibres and granuloma   shortening of the DDFT during healing by fibrosis.
           formation into the navicular bursa. Navicular bursi-  In some horses, heel elevation exacerbates lameness.
           tis may accompany the injury, with possible adhesion   The  outcome for  horses  with  primary  tendinitis
           formation between the dorsal surface of the tendon   treated with 6 months’ rest alone is disappointing,
           and the collateral sesamoidean and impar ligaments.   with only 25–30% returning to full athletic function
           The latter phases of healing are by fibrosis and the   and more than 60% suffering persistent or recurrent
           healing rate can be followed on MRI scans. On CT   lameness.  Several  additional therapies  have been
           images, tendon lesions may show focal hypoattenu-  tried, including injection of corticosteroids into the
           ation, enlargement of a tendon lobe and contrast   navicular  bursa  and/or  the  digital  sheath,  shock-
           enhancement in acute injuries.                 wave therapy, intralesional injection of biological
                                                          and regenerative products, inferior check ligament
           Management                                     desmotomy and bursoscopic debridement of dorsal
           The most important aspect of treatment is a long   surface tendon tears and granulomas (Figs. 1.212,
           period of rest (6 months or more). This should con-  1.213). So far, only bursoscopic debridement and
           sist of stall confinement with a low-grade mainte-  intrabursal/intrathecal corticosteroid therapy have
           nance daily exercise programme of 10–15 minutes   been evaluated critically. In a study of 92 horses with
           walking, starting after the first or second month   intrabursal tears, 42% were able to return to their
           of strict stall rest, depending on the severity of the   original level of performance following bursoscopic
           injury. Walking exercise can be increased gradu-  debridement of surface lesions of the intrabursal
           ally in duration but not intensity over the ensuing   portion of the DDFT, but 37% of horses suffered


           1.212                                          1.213























           Fig. 1.212  T2-weighted MR image showing dorsal   Fig. 1.213  Navicular bursoscopy showing a
           surface granulomas (arrows) on each lobe of the deep   surface tear in the deep digital flexor tendon with a
           digital flexor tendon.                         protruding disorganised mass of torn tendon fibres.
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