Page 480 - Atlas of Small Animal CT and MRI
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470  Atlas of Small Animal CT and MRI


              Figure 4.6.11  Eosinophilic Bronchopneumopathy (Canine)                                      CT
















             (a) DX, RLAT                     (b) CT, TP                       (c) CT+C, TP
             4y MC Rottweiler with 3‐month history of cough and recent progression to dyspnea and tachypnea, which has been unresponsive to
             cough suppressants and antibiotics. Radiographs revealed opacification of the accessory and dependent regions of the caudal lung lobes,
             consistent with consolidating alveolar infiltrates or pulmonary masses (a). Image b is representative of the caudal thorax in a lung
               window, and image c is a contrast‐enhanced image at approximately the same level as image b. CT images reveal pulmonary masses
             in the accessory and dependent regions of the caudal lung lobes (b: asterisks). Ground‐glass to alveolar infiltrates are also present in
             the nondependent regions of the caudal lung lobes (b: arrows). Patent airway lumina are evident within the masses in image b, and a
             complex grape cluster appearance in image C is indicative of additional thick‐walled, fluid‐filled airways incorporated within consoli-
             dated lung parenchyma. Lung biopsy revealed severe, diffuse, chronic eosinophilic bronchitis with eosinophilic granulomas. Microbial
             cultures were negative, and the dog responded to immunosuppressive doses of steroids but eventually developed severe bronchiectasis.
             (Same dog as in Figure 4.5.10 with end-stage bronchiectasis.)




              Figure 4.6.12  Endogenous Lipid Pneumonia (Feline)                                           CT





















             (a) CT, TP                       (b) CT, TP
             9y Domestic Shorthair with respiratory distress following thoracotomy and lung lobectomy for removal of a lung tumor. There is a diffuse
             reticulated interstitial to alveolar pattern throughout all lung fields (a,b). Average lung attenuation was approximately −250 HU. A right‐
             sided pneumothorax is present as a sequela of right caudal lung lobectomy (a). Postmortem examination revealed severe diffuse lipid
             pneumonia characterized by flooding of alveoli by large numbers of macrophages containing lipid droplets, confirmed by Oil Red O
             staining. There was also extensive pleural and septal fibrosis. Review of lung tissue from the lung lobectomy performed 6 days prior to
             postmortem examination showed no evidence of alveolar histiocytes. In this patient, the endogenous lipid pneumonia was thought to be
             associated with acute respiratory distress syndrome following thoracotomy.








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