Page 36 - Rapid Review of ECG Interpretation in Small Animal Practice, 2nd Edition
P. 36

Approach to Evaluating Arrhythmias





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               Fig. 3.4 Dog, lead II, 50 mm/s showing atrial fibrillation and f waves (arrow).



                              II




               Fig. 3.5 Dog, lead II, 50 mm/s showing atrial flutter and Fl waves (arrows).



          SUPRAVENTRICULAR ARRHYTHMIAS
          Supraventricular arrhythmias (SVAs) originate in   Most cases of AF in small animals are associated
          the sinus node, atrial tissue, and AV junction (or   with significant underlying heart disease. In giant
          AV node). SVAs include a wide variety of atrial, AV   breed dogs,  AF can occur spontaneously, and in
          junction, and  AV nodal tachycardias. SVAs have   the absence of identifiable heart disease, the term
          to be differentiated from sinus tachycardia. Sinus   “lone” AF is often used.
          tachycardia is typically physiological and may be
          caused by many conditions including febrile states,   Atrial flutter
          anemia, heart failure, adrenergic medications, and   AFL is a form of re-entry tachycardia that usually
          anxiety.                                       arises from the right atrium. It utilizes the anatomy
                                                         of the right atrium to sustain a macro-re-entry loop
          Specific supraventricular arrhythmias and ECG   of continuous depolarization. It can also be initiated
          criteria                                       and sustained in the left atrium. It is maintained
          Atrial fibrillation                            due to differences in the refractory periods of atrial
          AF is caused by multiple atrial impulses that   tissue. As in AF, the AV node controls the ventricular
          depolarize the atria simulatenously and bombard   response rate.
          the AV node. During AF, the atrial activation rate is
          rapid and can exceed 500 depolarizations/minute.   ECG criteria (Fig. 3.5):
          The refractory period of the  AV node does not     • Rapid ventricular rate possible.
          allow all these impulses to travel to the ventricle,     • Sawtooth undulation of the baseline.
          and prevents heart rates exceeding 250–300 bpm.     • Atrial (f wave) rate usually >300 bpm.
          This protects the ventricles from developing  VF     • Rate and regularity of RR intervals can be
          as a consequence of  AF.  The electrophysiological   variable (depends on AV conduction). Often 2:1
          mechanisms that initiate and maintain AF include   or 3:1 conduction patterns are observed.
          abnormal automaticity and re-entry.

          ECG criteria (Fig. 3.4):
             • P waves are absent.
             • Atrial activity is represented by fibrillatory (f)
            waves of varying amplitudes.
             • Irregular ventricular rhythm.
             • At very rapid rates the rhythm can appear more
            regular.
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