Page 856 - Adams and Stashak's Lameness in Horses, 7th Edition
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822   Chapter 7




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             A                                       B                          C

            Figure 7.15.  Radiographic images demonstrating incomplete   premature foal. Without support, normal weight‐bearing could lead
            ossification of the tarsal (A) and carpal (B) cuboidal bones in a   to wedging and collapse of the tarsal bones (C).

            careers than a group of normal foals.  Clinical signs
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            associated with cuboidal bone collapse may be evident
            in the newborn foal as an ALD of the carpus or tarsus.
            Collapse of the tarsal bones is much more common than
            that of the carpal bones. Tarsal collapse is often associ­
            ated with a sickle or cow‐hocked conformation of the
            tarsus or the tarsus looks like it has a “curb.” The degree
            of lameness is variable and may not become clinically
            apparent until later in life. Preventing cuboidal bone col­
            lapse in newborn foals with incomplete ossification
            involves minimizing compressive forces on the bones
            until they ossify. Confinement, sleeve casts, bandages, or
            bandages  and  splints  may  be  used  depending  on  the
            severity. In a previous study, foals with only minor tarsal
            bone collapse were able to perform as intended, whereas
            foals with more severe tarsal bone collapse and frag­
            mentation  could  not  be  used  for  their  intended  pur­
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            poses.  Additionally, incomplete ossification and mild
            collapse or wedging of the tarsal bones are thought to
            predispose to juvenile spavin in young horses. Horses
            with juvenile spavin have relatively severe signs of bone
            spavin at a young age with a history of minimal work
            (Figures 5.81, 10.27, and 10.58). Inherent abnormalities
            of the central and third tarsal bones are thought to con­
            tribute to the development of osteoarthritis (OA) in the   Figure 7.16.  Lateromedial radiographic view demonstrating a
            distal tarsal joints at such an early age. In those young   typical lateral ridge OCD lesion of the distal femur (arrow) and the
            horses  with obvious signs of juvenile  spavin, surgical   fragments that have migrated within the joint (arrowhead).
            arthrodesis is often effective in salvaging the animal for
            athletic use.

            Osteochondritis Dissecans (OCD)                    non‐weight‐bearing surfaces of the joint and are espe­
                                                               cially  common  in  the  stifle,  tarsus,  and  fetlock  joints
              OCD refers to cartilage or cartilage and bone (osteo­  (Figure  7.16).  Horses with OCD lesions typically
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            chondral)  fragments  or  flaps  that  develop  along  the   are only mildly lame but usually have joint effusion of
            articular surfaces of joints in horses. These abnormali­  the affected joint(s).  These lesions are often bilateral
            ties are probably the most common manifestation of the   and  may or may not require arthroscopic surgery to
            DOD complex in horses. They usually occur along the   remove the osteochondral fragment(s).  The prognosis
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