Page 228 - Feline diagnostic imaging
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13.14 Arteriotcromboembolism  231

                (a)                                                              (b)



























                (c)




                                                                        (d)















               Figure 13.28  A 6-year-old DLH presented for abdominal distension and decreased appetite. The cardiac silhouette was greatly
               enlarged on both the lateral (a) and ventrodorsal (b) images. Enlarged size of the caudal vena cava with pleural effusion and loss of
               abdominal detail due to fluid accumulation was apparent. M-mode of the right and left ventricle (c) shows severe enlargement of
               both ventricles. Fractional shortening was decreased to 13%. A right parasternal longitudinal axis view shows enlargement of the
               right atrium (d). Arrhythmogenic right ventricular cardiomyopathy was diagnosed.


               13.13   End-Stage Heart Failure                    now  are  thinned  due  to  progression  of  heart  disease.  On
               (Figures 13.29–13.31)                              presentation, fractional shortening may be low or normal,
                                                                  having dropped due to cardiac decompensation.
               Cats presenting in heart failure can be challenging to diag­
               nose and treat. Radiographic findings are not specific for the
               underlying cause as signs of left heart failure, biventricular   13.14   Arteriothromboembolism
               failure,  and  right  heart  failure  can  overlap.  Pulmonary   (Figures 13.32–13.35)
               edema is the most common finding with left heart failure;
               however, a percentage of cats can also present with pleural   Arteriothromboembolism  (ATE)  can  be  seen  associated
               effusion. Once heart failure occurs, the echocardiographic   with advanced cardiomyopathy, most commonly the hyper­
               findings may not fit neatly into the classic description of one   trophic form. The size of the left atrium regardless of the
               cardiomyopathy.  When  evaluating  a  patient  for  the  first   form of cardiomyopathy predisposes the patient to thrombo­
               time, you will not know if the walls had been thickened and   embolism.  Spontaneous  contrast  within  an  enlarged  left
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